Letting Kids 'Play Around' Is Actually Good For Them- Here's Why
When I met my friend Aishwarya's daughter, Aanya, on a Sunday afternoon, she was building an entire world in her backyard using only sticks, leaves, and her imagination. In her eyes, a stick became a magic wand, leaves turned into currency for her "shop," and an old cardboard box was her fortress. Aishwarya admitted that this was Aanya’s “free play” time—a period without gadgets, instructions, or adult guidance, which made me curious.
To put this to rest, we had a word with Dr Kushal Agrawal, Neonatologist and Head of the Department of Neonatology and Paediatrics at KVR Hospital Kashipur, "Unstructured play is much more than just frivolous fun. It is an integral part of child development. Today, children do not have much scope for free play. With the academic system, straddled by extracurricular activities and digital entertainment, the scope for unstructured play is getting smaller and smaller.". On the contrary, research reveals that unstructured plays hold a lot of benefits for children that structured activities may not have in store.
According to Dr Agrawal, "free time plays" are a great way for kids to get involved in physical activities meant for their growth. "When children are left to play freely, their movements become more varied and spontaneous," he says. Science behind this practice does support this because free play increases the levels of physical activity, which is moderately vigorous in children; this is essential in preventing childhood obesity and promoting health in general.
Other than these, the outdoor environments provide opportunities and challenges for natural development outside. While children run, jump, climb, and look around the outdoors, they can become able to develop strength, coordination, and endurance for a healthy and active lifestyle by exercising. Otherwise, structured activities often restrict children to specific movements or instructions.
Except for physical fitness, disorganized play lays the foundation for basic social and emotional capabilities. During unorganized play, children invent their games, negotiate roles, settle disputes, and collaborate to attain some common objectives. According to Dr. Agrawal, "such interactions build empathy, cooperation, and communication skills as children learn to express themselves, listen to others and adapt to group dynamics.".
Research indicates that children learn emotional resilience through unstructured play, navigating minor setbacks in a low-stakes environment. If the game goes sour, kids learn to solve the problem on the spot; this builds up their self-confidence as well as independence. This is a kind of play that gives children the freedom to learn by themselves and try new things without having the fear of being prohibited by others or the fear of failure.
Freedom in play helps unlock a child's creativity and the problem-solving aspects. Most structured activities have predetermined tasks or instructions that do not develop creative thought. According to Dr Agrawal, "When children are allowed to be free, they can explore and create their own scenarios, which are meant for developing unique solutions based on critical thinking.".
Unstructured play provides children with a number of materials for play—blocks, sand, or leaves, for example—without script, so that they invent games, build imaginary worlds, and, in a very real sense, answer their natural curiosity. It has been proven that this type of play helps children to make decisions, adaptability, and innovations, qualities useful throughout life.
Dr Agrawal believes that unstructured play is a very important factor of all-rounded child development. "Nowadays, children's lives are very scheduled, so it's very important for the parents to remember how necessary play is", he further adds. Being freely playful would be a great support to their healthy physical development as well as social, emotional, and cognitive development.
As parents, we can encourage free play by creating an environment that encourages exploration and curiosity. What can be very impactful is giving the child a safe outdoor space and setting aside gadgets to allow for uninterrupted time to play. As these spaces tend to be counterintuitive to everything that can be seen as a hallmark of productivity and measurable progress, unstructured play allows the children to build resilience, independence, and joy.
More than just being childhood, unstructured play confers a wide range of benefits since children learn to manage social dynamics, solve problems independently, and bring their creativity into life which puts them better prepared for the challenges of adulthood. In this natural form of play, people lay down a foundation for lifelong skills to achieve balanced wellbeing and success.
The next time you catch your child playing carefree—maybe using a stick as a wand or a rock as a pet—take the time to be thankful for the developmental steps.
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We may now have a clearer explanation behind ‘mommy brain’ or ‘momnesia’. Walking into a room and forgetting why you went there or losing track of a conversation is not simply a side effect of becoming a mother.
A new study has discerned a possible explanation for the phenomenon commonly known as “momnesia,” “pregnancy brain” or “mommy brain”. The findings were published in Science Bulletin.
According to the study, persistent high levels of estrogen during pregnancy may disrupt a particular brain circuit involved in memory.
Researchers found that high estrogen affected communication between the hypothalamus and hippocampus, two brain regions involved in regulating different aspects of brain function, including memory.
Pregnancy-related forgetfulness is generally considered to be temporary changes in memory and cognition during pregnancy.
Researchers say forgetfulness can become noticeable in late pregnancy stage and may continue well into the postpartum period before improving.
Women may have trouble recalling names or words, keeping track of events or completing everyday tasks.
According to the author of the study Dr Zheng Sun of Baylor College of Medicine, about 80% of pregnant women report these changes. The researchers say that this condition does not appear to be a decline in intelligence or cognitive ability.
According to Reuters, Dr Yanlin He, another study leader, said, “The important point is that pregnancy does not simply make someone ‘less intelligent.’ The effects appear to be more subtle and specific to certain cognitive tasks.”
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During pregnancy, estrogen levels increase drastically and reach its peak during the third trimester.
The researchers focused on estrogen receptor alpha in neurons located in the lateral hypothalamus. In experiments based on mice who were exposed to high estrogen level conditions like in pregnancy, these neurons became more active.
Those neurons send signals to the hippocampus, a brain region crucial for memory formation. The researchers found that increased activity in this region suppressed hippocampal activity and impaired performance on memory tests.
When researchers switched off the hypothalamus-to-hippocampus pathway, the memory problems in the mice were prevented. Activating the pathway, meanwhile, impaired memory even without elevated estrogen.
Removing estrogen receptors from the hypothalamic neurons also reversed the estrogen-related memory problems in the mice.
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The researchers also assessed memory in 70 women, including women at different stages of pregnancy and women who were not pregnant.
Memory problems surfaced during late pregnancy stages and were linked with higher circulating estrogen levels. The researchers reported changes across several memory-related tasks.
But it is important to note that the researchers did not directly measure or manipulate this specific brain circuit in pregnant women. That means the human findings support the mechanism observed in mice, but do not prove that exactly the same circuit causes memory changes during human pregnancy.
The study has cemented evidence that many pregnant women experience forgetfulness, but it does not mean pregnancy causes permanent memory loss. For most women, pregnancy-related forgetfulness generally improves after childbirth.
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The gut microbiome may influence how type 1 diabetes develops in children at high genetic risk, a new study suggests.
Researchers found that children whose gut microbiome stopped developing early had about three times the risk of developing type 1 diabetes or the immune attack that precedes it, compared with those whose microbiomes continued to mature.
The findings, published in Nature Metabolism, also showed that genetics influenced how strongly some microbiome patterns were associated with disease risk.
Type 1 diabetes affects more than 9 million people worldwide, including 1.8 million children and adolescents.
"Understanding the role of microbiome development in diabetes progression could lead to early prediction and prevention strategies, giving us more options to delay or even prevent the clinical manifestation of this disease," said co-corresponding author Daniel Wang, associate scientist with the Channing Division of Network Medicine in the Mass General Brigham Department of Medicine.
The longitudinal observational TEDDY Study followed 887 children at high genetic risk of type 1 diabetes and analyzed more than 12,000 stool samples collected during their first six years.
Participants came from Finland, Germany, Sweden and the United States.
Because the immune system can attack insulin-producing cells years before symptoms appear, researchers counted both the first detection of this immune attack and a clinical diagnosis as disease-related outcomes.
They identified three microbiome development patterns: early-matured, late-matured and early-plateaued.
Children with early-matured microbiomes developed greater bacterial diversity during their first year. Those with late-matured microbiomes started more slowly but eventually caught up.
The early-plateaued group showed slow development that failed to catch up, with low bacterial diversity persisting through the first three years.
Children with an early-plateaued microbiome had about three times the risk of developing type 1 diabetes or its preceding immune attack.
The association was also seen when the researchers analyzed the immune stage and clinical diagnosis separately.
The finding emerged from repeated stool sampling over time, suggesting a single microbiome snapshot could have missed the association.
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The bacterial differences also reflected how the children's microbiomes adapted as their diets changed.
In the early-matured group, bacteria shifted sooner from milk-adapted species such as Bifidobacterium toward bacteria that break down dietary fiber.
In the early-plateaued group, the microbiome remained more focused on digesting milk sugars even after solid foods were introduced and relied on a narrower range of bacterial species.
Lead author Danyue Dong, a postdoctoral research fellow in the Channing Division of Network Medicine at Mass General Brigham shared that "genetic background" can also influence the risk. Dong noted that combining microbiome and genetic information gives a more accurate picture.
"By analyzing interactions between the microbiome and host genetics, we found genetic variants, particularly those involved in antimicrobial and antiviral immune responses, that shaped how strongly the late-matured pattern was related to disease risk. The early-plateaued pattern, by contrast, carried higher risk regardless of genetic background," the expert said.
The study was observational, so it cannot establish that microbiome changes cause type 1 diabetes. Clinical trials are needed to determine whether modifying the microbiome can reduce disease risk.
The findings also came from children already at high genetic risk, meaning they may not apply to the general population.
Researchers say future pediatric care could potentially include microbiome testing during the first years of life. However, approaches such as dietary supplements would need to prove effective in clinical trials before being used for prevention.
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A baby girl who reportedly appeared healthy after being born died at just 14 months after getting afflicted with a devastating, ultra-rare genetic neurological disorder that made her incapable of moving, feeding and interacting with the world around her.
Poppy Massey was born in February 2022 with no signs or symptoms of any illness. Her mother Kaylee Massey's told PEOPLE that they began having concerns when Poppy was around four months old and her vision did not appear to be developing normally.
Further testing disclosed abnormalities in the corpus callosum, followed by diagnoses including microcephaly and cerebral visual impairment.
She eventually got diagnosed with TBCD leukodystrophy, a rare inherited disorder affecting the brain and nervous system. Poppy died on April 30, 2023.
TBCD disorder is a rare genetic condition that affects the brain and nervous system. It is caused by changes in both copies of a gene called TBCD, which is important for the normal development and functioning of nerve cells.
A child usually inherits one faulty copy of the gene from each parent. The parents typically do not have symptoms because they carry only one altered copy.
The condition can cause severe complications in brain development, movement, muscle strength and vision. Children may grow normally at first but then start showing symptoms like developmental delays, poor muscle, difficulties in feeding and moving, seizures, vision problems and loss of skills they had acquired previously.
TBCD-related neurodegenerative disease is sometimes grouped among leukodystrophies, a group of disorders that affect the brain's white matter.
White matter contains nerve fibres covered by a protective layer called myelin, which helps electrical signals travel efficiently through the brain and nervous system.
When this system is disrupted, communication between nerve cells can become impaired, contributing to the severe neurological symptoms seen in patients.
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One of the most difficult aspects of the disorder is that neurological problems may not be obvious immediately after birth. As the child's brain develops, symptoms can become more evident.
Brain imaging can show cerebral and cerebellar atrophy, a thin corpus callosum and impaired myelination. Some children subsequently experience stunted development.
In Poppy's case, her mother told PEOPLE that her condition deteriorated rapidly after diagnosis at nine months. She eventually lost the ability to suck and feed independently, lost movement in her legs and arms.
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The nervous system depends on organised cellular structures to develop, transport materials and communicate between neurons. Disruption of the TBCD protein can interfere with these processes during early brain development.
The disease involves brain atrophy and impaired myelination, affecting movement, development, vision, swallowing and other neurological functions.
TBCD is exceptionally rare. The TBCD Foundation says fewer than 50 diagnosed cases are known worldwide, although the true number may be higher because rare genetic disorders may largely remain undiagnosed.
Some affected children have very severe symptoms right from the onset of the disease disease, while others may survive longer. It varies depending partly on the specific genetic variants involved. Currently, there is no proven cure or treatment for TBCD disorder.
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