Winter is here, which means the season of sickness too is here. While viruses and sickness run throughout, winter is when the chilly weather further makes it worse for those prone to sickness. Especially, if they are kids, as their immunity is not fully developed.

Does that mean it is inevitable for kids to be sick?

Anytime a child is exposed to another child, they will fall sick. This is because germs are transferred in the air or by touch, and children, being curious, always touch things, including their own faces, mouths and easily pick up germs and spread them. While prevention is impossible, there are steps that can be taken to reduce this.

Experts and medical professionals recommend to follow the vaccine schedule by the Centers for Disease Control (CDC) and American Academy of Pediatrics (AAP). Vaccines have be proven to protect people, children, and infants from serious and deadly infections.

While sicknesses like a common cold do not have a vaccine, good hygiene can help prevent the spread of germs. Teaching your kids to wash hands after touching toys, or any other surface and other people is a great way to start it.

Exposure to germs at an early age strengthens child's immune system

Is there really any truth in the statement? When we encounter infections, our immune system creates antibodies that either prevent future infections or help the body fight them off more effectively, often leading to milder illness. The "hygiene hypothesis" proposes that living in an overly clean environment might hinder the immune system's development, potentially increasing the risk of allergic conditions like asthma.

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Although research has explored this idea, many immunologists (experts in the immune system) have raised concerns and expressed disagreement with the hypothesis.

In reality, most children don’t grow up in sterile environments. They are naturally exposed to a variety of germs, providing ample opportunities for their immune systems to strengthen and adapt.

Sanitizing Child's Toys

It is a good practice to regularly disinfect your children's toys and anything they share with others or take to school.

It is important to know the difference between cleaning, sanitizing, and disinfecting. While cleaning removes visible dirt, sanitizing decreases the number of germs on the surface, whereas disinfecting kills germs.

Also make sure to read the labels correctly and prevent any toxic toys to make its way to your kids, so it cannot be inhaled or ingested.

How to disinfect toys?

You can start by cleaning all the toys that has come in contact with your child's hands and mouth with soap and water. Then, use a sanitizer to clean everyday items, things that remain in your child's environment. Then use disinfectant, especially if your child is sick or if someone sick has touched your child's toys.

If you are wondering which sanitizers to go for, the Environmental Protection Agency (EPA) website has come up with a list of approved sanitizers and disinfectants that are effective.

Nearly 76,805 children and adolescents in India are estimated to develop cancer each year. At the same time, childhood cancer can be highly treatable when diagnosed early and managed appropriately, with survival exceeding 80% in settings where timely diagnosis and comprehensive treatment are available.

When people think about preventive healthcare for children, the conversation usually centers on vaccinations, good nutrition, physical activity, sleep and regular health check-ups. These are important for a child’s overall health. But prevention also involves something that is often overlooked: recognizing when a child’s health is changing and seeking medical advice at the right time.

Childhood cancer is different from many cancers seen in adults. In most cases, there is no clear lifestyle-related cause that parents could have avoided. A child developing cancer is therefore not usually linked to something the parents did or did not do. This is important because there is no particular diet, exercise routine or supplement that can guarantee protection against childhood cancer.

The Importance of Recognizing a Pattern

For doctors, one of the more practical ways to improve outcomes is to recognize when a seemingly common symptom is not following its usual course. Fever, tiredness, headaches, loss of appetite or body pain are frequent childhood complaints. But when they persist, keep returning or occur alongside other changes, they should not simply be treated repeatedly without evaluation.

• Unexplained weight loss

• Unusual bruising or bleeding

• Persistent bone or abdominal pain

• A growing lump or swelling

• Repeated headaches

• Changes in vision

These are some signs that may require further assessment. These symptoms are not specific to cancer, but their persistence warrants attention.

Why Early Diagnosis Matters

Most childhood cancers do not have a routine screening test for healthy children. Diagnosis therefore often depends on noticing an unusual pattern and investigating it appropriately.

A delay does not necessarily mean that cancer has been missed, but prolonged delays can allow some cancers to progress before treatment begins. For this reason, awareness among parents and healthcare providers, timely evaluation and appropriate referral are important parts of childhood cancer care.

Prevention Is Not Just About Avoiding Cancer

Vaccination still has a place in the broader cancer-prevention conversation. Hepatitis B vaccination helps reduce the risk of liver cancer linked to chronic hepatitis B infection, while HPV vaccination can help prevent HPV-related cancers later in life. However, these vaccines do not prevent most cancers that occur during childhood.

Similarly, nutrition should be viewed in the right context. A balanced diet supports growth and recovery, including during cancer treatment, but there is no specific food or diet that can prevent a cancer.

The Cost of Childhood Cancer

Childhood cancer can place a significant financial and practical burden on families. Treatment costs may be accompanied by expenses related to travel, accommodation, food and time away from work.

At the same time, families today have more avenues of support than before. Government health schemes, public healthcare facilities and non-government organizations can help with treatment costs and other practical needs. The challenge is ensuring that families are aware of these resources and can access them early enough.

Looking Beyond Treatment

Children who survive cancer may need long-term monitoring for possible effects of treatment on growth, development and other aspects of health.

For childhood cancer, therefore, prevention is less about finding a way to eliminate risk and more about reducing what can be changed: delayed diagnosis, interruptions in treatment and avoidable long-term complications..

(Dr Amita Mahajan, Senior Consultant, Pediatric Oncology, Indraprastha Apollo Hospital, Delhi)

Globally, one in six people of reproductive age experiences infertility at some point in their lifetime. Smoking is one of the key reasons for infertility among men and women worldwide, according to a new World Health Organization (WHO) knowledge summary.

The report, the 12th in a series of Tobacco Knowledge Summaries, highlights evidence linking tobacco use to infertility in women, harmful effects on sperm and sexual function in men, and potentially lower chances of success with some fertility treatments. It also warns that second-hand smoke may affect reproductive health.

Infertility is defined as the failure to achieve pregnancy after 12 months or more of regular unprotected sex.

What Does the WHO Report Say?

Infertility can have multiple causes. However, cigarette smoking and exposure to second-hand tobacco smoke are associated with infertility in women.

The harmful effects of cigarette smoking on women’s reproductive health are well documented, while evidence on other tobacco products, including waterpipe and smokeless tobacco, as well as nicotine products such as e-cigarettes, is still emerging.

Current data suggest potential risks to fertility, but further research is needed to fully understand these effects. Among

men, cigarette smoking is associated with harmful effects on semen and sperm, as well as sexual function, which can contribute to infertility.

On the other hand, women who quit smoking may have a lower risk of infertility compared with women who continue to smoke.

WHO recommends that healthcare providers inform individuals and couples planning pregnancy about the reproductive risks of tobacco use and offer evidence-based support to help them quit.

How Smoking Affects Fertility?

“Smoking is an important fertility consideration for both men and women. It can affect the quality of both eggs and sperm, and the concern is not limited to people who smoke themselves,” Dr Neelam Suri, Senior Consultant, Obstetrics & Gynecology, Indraprastha Apollo Hospital, Delhi, told HealthandMe.

Smoking also accelerates follicular depletion, which may contribute to an earlier onset of menopause compared with women who do not smoke.

Second-hand smoke can expose individuals to harmful substances that may affect reproductive health. Eggs and sperm are rapidly developing cells, and exposure to tobacco smoke can increase the risk of DNA damage and other cellular changes that may affect fertility and reproductive outcomes.

Dr Shalu , Senior Clinical Director, Cloudnine Group of Hospitals, Gurugram, told HealthandMe that while passive smoking may not directly cause infertility in every individual, regular exposure to tobacco smoke can affect reproductive health and have significant implications during pregnancy.

These include ectopic pregnancy, miscarriage, preterm birth, pre-eclampsia, fetal growth restriction, low birth weight, stillbirth, placental complications and neonatal complications.

Regular exposure to second-hand smoke may also potentially influence the chances of success with fertility treatments such as IVF.

“This is particularly relevant for couples undergoing IVF, where egg and sperm quality can influence the chances of successful treatment,” Dr Suri said.

What Should Couples Planning Pregnancy Do?

For couples planning a pregnancy or undergoing fertility treatment, avoiding tobacco smoke is an important step towards protecting reproductive health.

This means quitting smoking and maintaining a smoke-free environment at home to reduce regular exposure to cigarette smoke.

At the same time, fertility depends on multiple factors. Exposure to second-hand smoke does not necessarily mean a couple will experience infertility or IVF failure. However, reducing preventable risk factors can be beneficial when trying to conceive.

Ideally, couples should create a smoke-free environment before conception rather than waiting until fertility difficulties emerge.

Tylenol is one of the most commonly used pain killers. That’s why a study on the drug’s long-term effect on reproductive health of unborn girl children is concerning.

Researchers in Denmark found that girls exposed to paracetamol, also known as acetaminophen or Tylenol in the US, during fetal development had smaller ovaries, fewer ovarian follicles and smaller uteruses in infancy compared to girls who were not exposed to the component.

It’s essential to understand that study does not show that girls exposed to the drug will certainly have fertility problems later in life.

The researchers say it is still too early to know whether the changes observed during infancy and adolescence will translate into reduced fertility or earlier menopause.

What Did The Study Find?

Published in Human Reproduction Open, the study involved 302 girls from the Copenhagen Analgesic Study, or COPANA.

The researchers tracked mothers' paracetamol use during pregnancy through repeated questionnaires and also measured the drug in urine samples.

The girls were examined at around three months of age, during a period known as minipuberty, when the reproductive hormone system becomes temporarily active.

Of the 302 girls, 92 had first been exposed to paracetamol before 17 weeks of pregnancy; 67 were first exposed from 17 weeks onward and 143 had no reported prenatal exposure.

The researchers found that girls exposed during early fetal development had an approximately 40% smaller ovarian volume and a 13% smaller uterine volume compared to girls who were not exposed.

Girls exposed during mid-to-late fetal development stage had about 23% fewer ovarian follicles. These follicles contain immature eggs that can potentially develop and mature over a woman's reproductive life.

Additionally, girls exposed exclusively during early pregnancy also had lower levels of anti-Müllerian hormone (AMH), a hormone produced by ovarian follicles that is commonly used as a marker of ovarian reserve.

Also read: Tylenol, Other Medications Linked To 400% Rise In Liver Injuries In US

Changes From Infancy To Adolescence

Researchers also examined an independent group of 1,210 girls who had been followed from infancy through adolescence.

In this group, prenatal paracetamol exposure was associated with a smaller uterus at puberty and smaller ovaries during adolescence.

Study leader Margit Bistrup Fischer of Rigshospitalet in Copenhagen stressed that the long-term implications remain unknown. Whether these early reproductive differences affect fertility or the timing of menopause will require long-term follow-up.

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Tylenol And Infertility

This was an observational study, which means that researchers looked at an association between prenatal exposure to paracetamol and changes in future reproductive health.

They did not randomly assign pregnant women to take paracetamol, so the study cannot establish that the drug itself caused the changes.

The authors also say that external factors cannot be completely excluded. Women who took paracetamol because they must have been pain and discomfort due to headaches, fever or other illnesses. These underlying conditions could also influence fetal development.

Also, the women used relatively low to moderate amounts of paracetamol. According to COPANA cohort, the most common reasons were headaches or migraines, musculoskeletal pain, illness or cold and fever.

The study also found that increased reported paracetamol exposure, meaning high dose, was too associated with lower ovarian and uterine volumes.

Also read: Paracetamol Is Not Safe For Older People, Finds Study

Earlier Studies That Raised Similar Concerns About Tylenol

It's not the first time that Tylenol has sparked safety concerns. In 2025, new studies by Mount Sinai and Harvard T.H. Chan School of Public Health raised an concerning question: is prenatal exposure to acetaminophen associated with neurodevelopmental disorders including autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) in children?

A study led by Andrea Baccarelli, dean of Harvard T.H. Chan School of Public Health’s faculty of environmental health in BMC Environmental Health, systematically reviewed 46 previous studies encompassing data from more than 100,000 participants.

The aim was to investigate whether acetaminophen use during pregnancy correlates with neurodevelopmental disorders in children.

Applying the Navigation Guide Systematic Review method—a strict, gold-standard approach to assessing environmental health evidence—the study team evaluated the quality, risk of bias, and strength of evidence of all included research.

By doing so, it was possible to conduct a solid synthesis of available scientific literature, and what emerged was a pattern: better-quality studies showed a more pronounced association between prenatal acetaminophen exposure and offspring increased risk of ADHD and autism.

Another recent study observed that liver injuries reported to US poison centres increased by nearly 400% between 2000 and 2024, due to acetaminophen, sold under brand names like Tylenol.

The study also found that exposures involving acetaminophen-containing combination drugs decreased by 60%-85% after the US Food and Drug Administration capped the amount of acetaminophen allowed in combination prescription products.

However, harmful exposures to acetaminophen alone increased steadily over the 24 years. The findings suggest that regulatory action reduced liver injuries linked to combination products, while acetaminophen alone remained a leading contributor to poison-centre-reported liver injuries.