(Credit-Canva)
Feeling sleepy after eating a big meal or after an early morning is normal. While excessive sleepiness does raise alarm, the key to avoid it to understand what is triggering it.
Our bodies have an internal clock, called the circadian rhythm, that controls when we feel sleepy or awake. According to National Institute of General Medical Sciences (NIGMS), these rhythms include physical and mental changes an organism experiences over 24-hour cycle.
According to John Hopkins Medicine this clock makes us naturally feel a bit sleepy in the early afternoon, usually around 1:00 or 3:00 PM. It's like a built-in dip in our energy levels. Scientists have studied this, and they've found that we're usually most alert in the morning and early evening, with a little dip in between. In some countries, people used to take a short nap in the afternoon to deal with this. It's just a normal part of how our bodies work, but we can learn how to manage it.
Everyone gets sleepy in the afternoon sometimes. It's a normal part of life. But you don't have to let it ruin your day. Sometimes, feeling really sleepy during the day can be a sign of a medical problem. If you have a condition like sleep apnea or narcolepsy, or if you take medicine that makes you sleepy, you might feel extra tired in the afternoon. The natural dip in alertness makes it even worse for you. If you're feeling excessively sleepy during the day, it's important to talk to your doctor. They can help you figure out what's going on and find ways to feel more awake and alert. By taking care of your sleep habits, you can make it easier to get through the day without feeling tired all the time. Here are some ways you can avoid the afternoon crash.
Quality sleep is crucial. Aim for consistent bedtime routines and sufficient hours of rest. Sleep deprivation amplifies afternoon fatigue. A well-rested body manages energy dips more effectively, reducing the severity of the midday slump.
Even brief movement combats sleepiness. Stand up, stretch, or take a short walk. Physical activity boosts blood flow and oxygen to the brain, enhancing alertness. Regular, light activity throughout the day helps maintain energy levels.
Heavy, carb-loaded lunches divert energy to digestion, inducing sleepiness. Opt for balanced meals with lean protein and vegetables. Lighter meals prevent excessive energy shifts, helping you stay alert and focused in the afternoon.
Moderate caffeine intake can temporarily increase alertness. However, avoid excessive amounts, especially later in the day. Hydration is vital; water improves circulation and cognitive function. Choose refreshing drinks to stay hydrated and energized.
Vitamin C's antioxidant properties may reduce fatigue. It combats oxidative stress, a contributor to tiredness. Consider a supplement or vitamin C-rich foods. Remember, individual responses vary, and it's best to consult a healthcare professional.
When feeling sleepy, change your mental environment. Focus on completed tasks or engage in enjoyable activities. Distraction can redirect your mind, temporarily alleviating the feeling of sleepiness. A brief mental break can provide a needed reset.
Short naps (20-30 minutes) can rejuvenate, but avoid longer naps. They can lead to grogginess. Napping too close to bedtime disrupts nighttime sleep. A brief nap during the early afternoon can restore alertness and improve productivity.
Credit: Naga Munchett/ Instagram
The BBC Breakfast host Naga Munchetty has opened up about her experience of suffering from a painful womb condition, known as the "evil twin sister of endometriosis".
The 51-year-old Naga Munchetty explained her condition as adenomyosis, which can cause extreme pain at any time. Munchetty added that she has faced the condition even while presenting her show, The Independent reported.
Naga Munchetty said that adenomyosis has caused her severe pain since she was in her teens, and she has “become conditioned to accept” it.
“If you’re curled up on the floor screaming, sweating, flooding, passing out, vomiting, that is debilitating. But you end up normalizing that pain.”
Adenomyosis is an extremely painful condition, which affects the womb and causes the uterus to enlarge. Although it affects an estimated one in 10 women, it remains undiagnosed in most women.
It is a lesser-known but significantly debilitating gynecological disorder that is commonly mistaken for endometriosis.
Adenomyosis leads to debilitating symptoms such as
Adenomyosis occurs when endometrial cells—typically restricted to the lining of the uterus—break through the myometrium, the muscular wall of the uterus.
Endometriosis, on the other hand, happens when tissue similar to the lining of the uterus grows outside the uterus. These tissues commonly develop on the ovaries, fallopian tubes, or the pelvic lining.
In contrast, adenomyosis is inside the uterus but produces serious complications. These out-of-place endometrial cells continue to act as they would in a regular menstrual cycle, becoming thick and shedding, which leads to internal bleeding, inflammation, and intense pain.
Despite going to the doctors several times, Naga Munchetty was not diagnosed until recently, as she was led to believe the pain she was experiencing was “normal”.
No absolute cure for adenomyosis exists except for a hysterectomy, which involves the removal of the uterus. Nevertheless, several treatment options can alleviate symptoms and enhance quality of life. These include:
1. Pain Relief
Ibuprofen and naproxen, over-the-counter pain medications, are usually prescribed to relieve menstrual cramps and pain.
2. Hormonal treatment such as
GnRH agonists, such as Lupron, induce temporary menopause by suppressing estrogen production, thereby reducing adenomyosis symptoms. However, these injections can cause severe side effects, including mood swings, hot flashes, and bone density loss, making them unsuitable for long-term use.
4. Surgical Intervention
According to experts, the brain is responsible for high BP. (Photo credit: iStock)
Hypertension, or high blood pressure, is caused by excessive salt intake; however, according to scientists, there are more reasons. Experts also blame a brain glitch. A part of the brain that makes us breathe while laughing or coughing could also cause a blood pressure spike, as per researchers from New Zealand and Brazil. This could explain why medication is not enough for controlling BP in some people. According to experts at the University of Auckland, there is a part of the brain that could cause high BP, thereby suggesting that the brain is responsible for hypertension.
Experts say that the lateral parafacial region is located in the brainstem. It is the oldest part of the brain and regulates heart rate, digestion, and breathing. Its action causes one to exhale during a cough, laugh, or workout. These actions are driven by abdominal muscles. Researchers also found that this area could connect to the nerves that tighten blood vessels and raise blood pressure. However, it is possible to reverse this action by inactivating this region.
Hypertension, or high blood pressure, is a condition wherein blood pushes against the walls of the blood vessels with too much force. Initially, it has no symptoms, but it is a silent killer. Over time, untreated high BP can lead to a stroke or a heart attack. While it is mostly believed to be a consequence of smoking, drinking alcohol, eating salty foods, and not exercising, experts have found other causes as well. As per research by the University of São Paulo in Brazil and the University of Auckland in New Zealand, published in the journal Circulation Research, rat brains had their brain cells in the lateral parafacial region switched off.
Researchers in the study successfully reactivated neurons in some rats and found that brain circuits ultimately raised BP. Experts mapped out what happened and compared it with rats that did not have hypertension. In the hypertensive group, lateral parafacial region neurons were helping with breathing but were also raising BP. This suggests that changes in breathing, which involve abdominal muscle contractions, can also trigger high BP. This is why patients with sleep apnoea experience interrupted breathing while sleeping and high blood pressure.
Apart from normal breathing, lateral parafacial region neurons are activated when oxygen levels are low. Experts also checked whether they could trigger the brainstem with medication. They noted that it was tricky because drugs would work on the entire brain and not a specific region. It could be activated by signals from the neck cells near the carotid artery. These can be safely targeted with medication.
To control persistent high BP, medication every day is a must. But to intensify the benefits, try the following simple tricks:
Drinking water and hydrating properly can help you dodge kidney problems. (Photo credit: iStock)
Chronic kidney disease (CKD) is no longer a distant public health concern—it is something we are seeing in clinics every day. With over 600 million people affected globally and a rapidly increasing burden in India, the usual culprits—diabetes and hypertension—are well established. However, in routine practice, one factor that is consistently overlooked is chronic underhydration.
Dr Anurag Gupta, Senior Consultant Nephrologist and Dialysis Director at Sir Ganga Ram Hospital, in an interview with Health and Me, spoke about the consequences of dehydration on the kidneys.
In Indian settings, especially during summers, it is not uncommon to see patients—drivers, outdoor workers, even hospital staff—going long hours with minimal fluid intake. Many of them present with recurrent stones, urinary infections, or unexplained decline in kidney function. While we may not always label dehydration as the primary cause, it is often a silent contributor.
From a physiological standpoint, the explanation is straightforward. Reduced fluid intake leads to lower renal perfusion and higher vasopressin levels, resulting in concentrated urine and increased tubular workload. When this becomes a daily pattern rather than an isolated event, it is reasonable to believe that cumulative injury occurs. Emerging data from heat-stressed regions, including parts of India, are now supporting this clinical observation.
Another issue is the widespread misunderstanding around hydration. Some patients barely drink water through the day, while others—after a diagnosis—swing to the opposite extreme, assuming excessive water intake will “clean” the kidneys. Both approaches are problematic. Overhydration, particularly in the elderly, can lead to hyponatremia, something we do encounter in practice. What works better is simple, practical advice—maintaining a light-coloured urine is often more useful than prescribing a fixed number of litres.
At the same time, hydration cannot be viewed in isolation. Our dietary and lifestyle patterns are equally concerning. High salt intake remains the norm rather than the exception, directly contributing to hypertension and kidney damage. Sedentary lifestyle and poor glycemic control further compound the problem. Another area we tend to underestimate is the indiscriminate use of over-the-counter painkillers and alternative medications. Many patients do not even consider these harmful, yet we frequently encounter kidney injury linked to them.
Protein intake is often a source of confusion, particularly with the rising popularity of high-protein diets. In otherwise healthy individuals, moderate intake is not an issue. However, in CKD patients, unchecked protein consumption can accelerate disease progression. On the other hand, excessive restriction—sometimes self-imposed—can lead to malnutrition. This is where individualised guidance becomes essential, rather than blanket advice.
In day-to-day nephrology practice, doctors often focus heavily on pharmacological management. While necessary, it should not overshadow basic preventive measures. Adequate hydration, reduced salt intake, sensible protein consumption, and avoidance of nephrotoxins are simple interventions, but they are not emphasised enough.
If we are serious about addressing the growing CKD burden in India, these “basic” measures need to be brought back to the center of our conversations—with patients and within the medical community.
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