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Sleep changes as we age. When we were kids, we needed 10 to 12 hours of sleep, for teens it is eight to 10 and it decreases to seven to nine as we reach adulthood. But there is always a lack of urgency when it comes to sleep and young adults, many of whom prefer staying up and doing different activities. According to the Centre of Disease Control and Prevention, many national surveys show that about 37% of men, and 39% of people from the age of 45 to 64 reported not getting enough sleep.
Many people in America don't sleep the right amount. This means they either sleep too long or not long enough. But what happens to your body when you do not sleep enough? You just feel tired right? No, when you don't get this much sleep, your body can get stressed. This can make you more likely to get sick. A study published by the JAMA Network Open Sleep Trajectories and All-Cause Mortality Among Low-Income Adults showed that people who don't sleep the right amount have a higher chance of dying early. It's like your body needs that time to rest and fix itself. Without enough good sleep, things can start to go wrong. So, getting the right amount of sleep is super important for staying healthy.
Scientists wanted to see how sleep habits affect people's health over many years. They looked at almost 47,000 people who were between 40 and 79 years old. They asked them about their sleep habits when the study started, and then again, a few years later. The scientists wanted to see if people's sleep habits changed. They divided people into groups based on if they started with too much or too little sleep, and if their sleep changed over time. For example, some people started sleeping a lot but then started sleeping very little. This helped the scientists see how different sleep patterns affected people's health. They wanted to see the long-term effects of sleep.
The study found that people who had sleep habits that changed a lot had a higher risk of dying early. This means if you started sleeping too much and then switched to sleeping too little, or the other way around, you were more likely to die sooner. They also found that these people had a higher risk of heart problems. The risks were even higher for some groups of people, like white adults and people with higher incomes.
If you often doze off when you are sitting and reading, watching a movie, talking to someone, sitting quietly after lunch or even during a few minutes of traffic, you may be sleep deprived according to the National Heart, Lung, and Blood Institute. They explain how sleep deprivation can cause issues with learning, focusing and reacting to certain things. The symptoms of sleep deprivation in kids differ a little as they might be overly active and have trouble paying attention to certain things. If you are experiencing sleep issues, make sure to speak to a healthcare professional who will help you identify the issues and direct you towards the treatment or changes you must make. Here are some ways sleep helps your body.
Good sleep allows your heart and blood vessels to heal. This keeps them strong and healthy, reducing the risk of heart problems.
Sleep helps balance your hunger hormones, so you don't feel too hungry. This helps prevent eating too much and keeps your weight healthy.
Proper sleep helps your body use insulin correctly. This lowers the chance of high blood sugar, which can lead to diabetes.
Deep sleep releases growth hormones, helping kids and teens grow. It also repairs body tissues, which is important for everyone.
When you sleep well, your body's defense system gets stronger. This helps you fight off germs and stay healthy.
Credit: AI generated image
Climate change caused a 10 per cent global increase in Salmonella antibiotic resistance genes between 1940 and 2023, according to the first-of-its-kind study published in The Lancet Planetary Health journal today.
Antimicrobial resistance (AMR) is mainly driven by the overuse and misuse of antibiotics, which allows resistant bacteria to survive and spread.
However, rising temperatures and changing rainfall patterns can influence how bacteria survive, mutate, and spread, potentially increasing the exchange of antibiotic resistance genes.
“The accumulated evidence suggests that climate change is an accelerating force behind the global spread of antimicrobial resistance,” the study authors wrote in the paper.
The study provides supporting evidence that AMR doesn’t just increase steadily as temperatures rise, but that the number of resistance genes changes over time in a more complicated way, depending on both temperature and rainfall. This suggests that environmental changes can speed up how bacteria adapt to antibiotics.
“These findings reinforce the idea that climate change alters microbial ecological stability and accelerates resistance evolution across human, animal, and environmental reservoirs," said the global researchers.
The current study analyzed the genomes of more than 480,000 Salmonella samples from 139 countries, collected between 1940 and 2023, and compared levels of antibiotic resistance genes with changes in average temperature and rainfall over time.
Of the total, 82 per cent of countries saw increases in antibiotic resistance genes in Salmonella, with the strongest climate-associated increases occurring in the Middle East and North Africa, followed by South Asia and Sub-Saharan Africa.
While the study shows a link between climate change and antibiotic resistance genes in Salmonella, it does not prove that climate change directly causes the increase.
The study also used a model to predict the change in antibiotic resistance genes in Salmonella by 2100 under different climate emissions scenarios.
The model suggests that if countries meet low-emission climate targets and strengthen efforts to use antibiotics responsibly, levels of resistance genes could be 24% lower than under the highest-emission scenario. However, they caution that these projections, as with all models, involve uncertainty.
The researchers stressed the need to consider climate change when monitoring and addressing AMR. They add that stronger climate action, alongside responsible antibiotic use and improved disease surveillance across humans, animals, and the environment, will be important in limiting the future spread of AMR.
As per the US Food and Drug Administration (FDA), Salmonella is a group of bacteria that can cause gastrointestinal illness and fever called salmonellosis. It can be spread by food handlers who do not wash their hands and/or the surfaces and tools they use between food preparation steps. It can also happen when people consume uncooked and raw food. Salmonella can also spread from animals to people.
Common symptoms of Salmonella include
Children younger than 5, adults 65 and older, and people with weakened immune systems are more likely to have severe illness.
Heat already pushes the body to its limits; smoking removes its safety net. (Photo credit: AI generated)
Indian summers are not just uncomfortable; they are becoming increasingly dangerous. With temperatures frequently crossing 45–48°C, heatwaves are putting excess stress on the human body, which hitherto had not experienced this level of heat strain. Now, add smoking to this already hostile environment and, like adding fuel to a fire, two harmful components combine to multiply the damage. Dr Shubham Garg, Director of Surgical Oncology, Dharamshila Narayana Superspeciality Hospital, Delhi, spoke about the risks of stepping out to grab a smoke during extreme heatwaves.
Smoking during heatwaves doesn’t just worsen existing risks; it accelerates dehydration, strains the heart, damages the lungs, and pushes the body closer to heat exhaustion or heatstroke. Here’s why lighting up in extreme heat is far more dangerous than most people realise.
When temperatures soar, your body works overtime to cool itself. A host of processes happen to aid in this—your blood vessels dilate, there could be an increase in heart rate, and sweating intensifies in order to regulate body temperature. When you smoke, it interferes with these very natural defense mechanisms of your body.
Nicotine results in vasoconstriction—narrowing of blood vessels—which makes it very difficult for the body to release heat trapped inside. The carbon monoxide from cigarettes reduces oxygen delivery to tissues. The result? Less oxygen reaches your organs, which are, in fact, working harder in the extreme heat. This is a perilous combination that can affect the body in many ways.
A heatwave leads to sweating and, consequently, loss of fluids and electrolytes. And when you go for smoking a cigarette, it leads to fluid loss and delayed hydration. Nicotine acts as a mild diuretic, which contributes to increased fluid loss. Smoking also suppresses thirst signals, thus delaying hydration.
Collectively these factors raise the risk of severe dehydration, which can trigger dizziness, muscle cramps, low blood pressure, and confusion—all of which are early signs of heat exhaustion. Many smokers ignore these signs or dismiss them altogether.
Cardiovascular strain can happen independently through either smoking or heat. That in itself is a threat one should keep an eye out for. However, when combined, they pose a compelling risk of:
During extremely hot weather conditions, especially during a heatwave, the heart has to exert more effort to maintain circulation and cooling in the body. Smoking elevates heart rate and blood pressure further while also thickening the blood and increasing the risk of heart attacks and strokes, especially in people with pre-existing diabetes, hypertension, or heart disease.
Hot weather is bad for air pollution levels too, as it traps smoke, dust, and harmful gases close to the ground. When one smokes in these conditions, it severely compromises lung function:
For people with asthma, COPD, or other respiratory conditions, smoking during a heatwave is likely to trigger severe flare-ups and emergency hospital visits.
Extreme heat is damaging not just for the heart but for the skin as well. The skin becomes dehydrated, and collagen breaks down. Smoking compounds this damage by reducing blood flow and oxygen supply to the skin.
The result:
In short, smoking during summer doesn’t just harm internal organs; it visibly accelerates the ageing process.
Smoking reduces the body’s ability to regulate temperature effectively. This makes smokers more vulnerable to heat exhaustion (fatigue, nausea, headache, dizziness) and heatstroke (confusion, collapse, organ failure).
Heatstroke is a medical emergency and can be fatal if not treated promptly. Smokers often misread early warning signs as ‘normal summer weakness,' thus delaying care.
Many smokers try to “reduce” smoking during summer. While any reduction helps, heatwaves are one of the worst times to smoke at all. Even a few cigarettes can significantly increase physiological stress when temperatures are extreme.
Smoking during heatwaves is not just bad—it’s dangerously synergistic. If there ever is a time to quit, or at least pause, this should be it. Because in peak summer, smoking doesn’t just harm you slowly. It fast-tracks damage, turning heat into a silent but serious health threat. In extreme heat, choosing not to smoke isn’t just a lifestyle choice—it’s a life-saving one.
Even imperfect CPR is better than no intervention at all. (Photo credit: AI generated)
When the heart stops functioning, time doesn’t stop with it. In cases of cardiac arrest, time serves as one of the most decisive factors between survival and irreversible loss. Within a couple of seconds, the body starts losing its oxygen supply. In a few minutes, the brain starts to suffer damage. And with each passing minute without intervention, the chances of survival reduce significantly.
This severe reality is at the centre of what Dr Ankit Desai, Paediatric Anaesthetist and Founder & Director of Children’s Anaesthesia Services, explains as “a race against biological shutdown — one where the bystander is the only lifeline”.
Several people have the misconception that cardiac arrest is similar to a heart attack, but they are very different. A heart attack is a circulatory issue where the heart might still be beating. However, in cases of cardiac arrest, there is an electrical failure, and the heart suddenly stops pumping blood effectively.
Whenever this occurs, blood flow to the brain and other vital organs ceases immediately. The oxygen reserves in the brain are extremely limited and typically last for about 4 to 6 minutes before any permanent injury occurs.
This is where the concept of time sensitivity becomes more important. For every passing minute without CPR or defibrillation, the chances of survival drop by approximately 7–10%. By the time 10 minutes have elapsed without intervention, survival is extremely unlikely in most cases.
“The tragedy is not just the cardiac arrest itself,” explains Dr Desai, “but the silence that follows — when no one knows what to do or hesitates too long to act.”
The brain is the first organ to be affected during cardiac arrest. Neurons are highly sensitive to oxygen deprivation. Brain cells start to malfunction within 3 minutes. By 5 minutes, the damage starts becoming increasingly severe. Beyond 10 minutes, the chances of meaningful recovery drastically reduce. This is why immediate CPR is not just a supportive measure but a bridge that keeps oxygen flowing artificially until a normal rhythm can be restored.
Chest compressions manually pump blood to the brain and heart, delaying cell death.
Emergency medical services, even in well-equipped systems, often take several minutes to reach a patient. In urban areas, response times may be shorter, but they are rarely instantaneous. In cardiac arrest, those minutes matter more than any hospital intervention.
Dr Desai emphasises that “the first responder is almost always not a doctor — it is a family member, a colleague, or a nearby stranger”.
This makes bystander CPR the most critical determinant of survival. Studies consistently show that when CPR is initiated immediately, survival rates can double or even triple compared to cases where no bystander action is taken.
Yet fear, hesitation, and lack of training remain major barriers. Many people worry about performing CPR incorrectly, causing harm, or being held legally responsible. In reality, doing nothing is far more dangerous than taking imperfect action.
Medical professionals often refer to this situation as the “Chain of Survival”, which includes early detection of cardiac arrest, immediate CPR, rapid defibrillation (AED use), advanced medical care, and post-resuscitation support. Every link in this chain is highly time-sensitive. Any delay in one step weakens the entire outcome. The strongest determinant, however, remains the second step — early CPR.
Automated External Defibrillators (AEDs), if available, can help restore a normal heart rhythm if used quickly. But again, their effectiveness decreases sharply with delay. The combination of CPR and early defibrillation within the first few minutes offers the best chance of survival.
The key difference between life and death is less about complexity and more about readiness.
Awareness training helps transform bystanders into responders. A person who knows how to identify cardiac arrest — unresponsiveness, absence of breathing, sudden collapse — is far more likely to act immediately rather than wait.
Dr Desai highlights a critical cultural gap: “We often associate medical emergencies with hospitals. But cardiac arrest begins in living rooms, offices, gyms, and streets. The response must begin there, too.”
Basic CPR training takes less than an hour to learn, but can influence outcomes for decades. Schools, workplaces, and community programmes play a vital role in normalising this skill.
One aspect of cardiac arrest that often gets overlooked is human hesitation. Bystanders often freeze due to shock and uncertainty. Some assume that someone else will step in. Others underestimate the severity of the situation.
Public awareness campaigns help highlight the simplicity of CPR, which helps overcome this barrier. Hands-only CPR focuses on continuous chest compressions without mouth-to-mouth breathing, making intervention much easier and more accessible. The message is simple: push hard, push fast, and don’t stop until help arrives.
Cardiac arrest survival is not just a medical issue, but also one of public preparedness. The Chain of Survival starts long before the emergency happens. It starts with education, confidence, and awareness.
Dr Desai states that “if more people understood how little time they truly have, more lives would be saved not by hospitals, but by ordinary people doing extraordinary things in the first five minutes”.
Conclusion: time is the real patient
In cardiac arrest, the patient is not just the person who collapses — it is time itself. Every second lost reduces the chance of recovery. Every trained bystander becomes a potential lifesaver. The science is clear, the timeline is unforgiving, and the solution is remarkably simple: act immediately, compress the chest, and keep blood flowing until professional help arrives.
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