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Many people believe that the modern-day skincare trends are a scam made by the big pharma companies who wish to make more money. This is a recent trend of ‘Anti-Aging’ skincare that keeps yourself on toes for the innate need for women to stay ‘young’ if they wish to be desirable. The beauty industry, in a way, feeds on such societal-made insecurities. But the recent rise in the trend has taken a different turn. While before people did it before to look prettier or fairer, now people are looking at this skincare routine as a way of taking care of themselves rather than doing to match someone else's beauty standards. This is why, this trend has resurfaced in the skin care community. This is the blue light routine.
In today's digital age, where we spend countless hours looking at screens, from phones and tablets to laptops. This constant screen time exposes us to blue light, which research suggests can have negative effects on our skin. It also leads to premature aging and dark spots. The screens also leads to stress, which could further cause harm to skin. While ditching our devices isn't realistic, a growing number of skincare products claim to protect against blue light. But do these products actually work?
You can find many skincare products that say they protect against blue light, like sprays, creams, gels, and sunscreens. Some promise to undo the damage blue light might cause, while others try to prevent it in the first place. Blue light sunscreens are special because they protect against both the sun's UV rays and blue light. This is what regular sunscreens don't do very well. Regular sunscreens, whether they use chemicals or minerals, don't block blue light as effectively.
However, tinted sunscreens are different. Tinted sunscreens with SPF 30 or higher can protect your skin from blue light, as well as UVA and UVB rays from the sun. This is because of the coloring in the tint. Some newer sunscreens also have special ingredients that help protect against visible light, including blue light. While scientists are still learning about how blue light affects skin, many skin doctors have found it to be helpful for skin.
Blue light comes from screens, TVs, and even the sun. While we used to worry mostly about how it affects our eyes and sleep, now we're learning it might also affect our skin. Some studies show that too much blue light can damage skin cells and speed up aging, leading to wrinkles and uneven skin tone. It can also cause dark spots on the skin. This happens because blue light can cause something called "oxidative stress" in the skin, which is linked to aging. It leads to dead skin cells. Blue light goes deeper into the skin than the sun's UV rays and can make skin cells produce more pigment, causing dark spots. It's important to know that not all blue light is bad. There is a difference between the blue light that comes out of the screens and the blue light that is used from a certain blue light wavelength as a therapy. The latter is used by doctors to treat acne and some skin cancers.
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Can Alzheimer's be completely reversed? This is not just about preventing it or ensuring it that the disease slow down, but can it be reversed to achieve full neurological recovery? For the longest, we have known that Alzheimer's is a progressive, degenerative brain disease, which destroys memory, thinking, and eventually the ability to perform simple tasks, but now a team of researchers from Case Western Reserve University, University Hospitals (UH), and Louis Stokes Cleveland VA Medical Center has challenged this belief. They may have found out something that could reverse it, at least so says the animal model.
Instead of targeting plaques or tangles alone, the team looked at something more fundamental: the brain’s energy system.
Their findings, published in Cell Reports Medicine, suggest that restoring the brain’s energy balance may not just slow Alzheimer’s but potentially reverse key features of the disease, at least in animal models.
The study is led by Kalyani Chaubey from the Pieper Laboratory, and at the center of the study is NAD+, a molecule essential for cellular energy and repair.
NAD+ levels naturally decline with age across the body, including in the brain. When levels drop too low, cells struggle to perform basic functions and eventually fail.
The researchers found that this decline is far more severe in the brains of people with Alzheimer’s. The same sharp drop was also seen in mouse models of the disease, pointing to a shared biological problem.
While Alzheimer’s is uniquely human, scientists use specially engineered mice to study it. In this study, two types of mice were used. One model carried human mutations linked to amyloid buildup, while the other carried a mutation affecting the tau protein.
Both amyloid and tau are central to Alzheimer’s pathology. Over time, these mice developed symptoms similar to human Alzheimer’s, including brain inflammation, damage to nerve fibers, breakdown of the blood-brain barrier, reduced formation of new neurons, and severe memory and learning problems.
After confirming that NAD+ levels were dramatically reduced, the researchers tested whether restoring this balance could help. They used a drug called P7C3-A20, developed in the Pieper Laboratory, which supports cells in maintaining healthy NAD+ levels under stress.
Remarkably, the results went beyond prevention. Even when treatment began after significant disease progression, the mice showed reversal of major brain damage. Cognitive function fully recovered in both mouse models, despite their different genetic causes.
The recovery was not just behavioral. Blood levels of phosphorylated tau 217, a biomarker now used clinically in humans to detect Alzheimer’s, returned to normal in treated mice. This provided objective evidence that disease processes had been reversed, not merely masked.
The findings suggest a possible paradigm shift. Alzheimer’s damage may not always be permanent. Under certain conditions, the brain appears capable of repairing itself and regaining function.
However, the researchers caution against self-medicating with over-the-counter NAD+ supplements. Some have been shown in animal studies to raise NAD+ to unsafe levels that may increase cancer risk. The drug used in this study works differently, supporting balance rather than excess.
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A newly recognised form of diabetes is reshaping how scientists and doctors view the condition, particularly in countries like India. In 2025, global health authorities officially acknowledged Type 5 diabetes as a separate and distinct form of the disease.
This recognition ended decades of confusion around a type of diabetes that did not fit neatly into the existing categories of Type 1 or Type 2 diabetes. The formal classification, backed by the International Diabetes Federation and supported by research published in The Lancet Global Health, is expected to transform diagnosis, treatment, and long-term care for millions of people worldwide.
But what exactly is Type 5 diabetes, and how does it differ from the types of diabetes that are more widely known?
Type 5 diabetes is now recognised as a distinct form of the disease caused primarily by severe, long-term malnutrition, often experienced during childhood. This undernutrition can lead to profound insulin deficiency and an underdeveloped pancreas, resulting in significantly reduced insulin production.
Unlike Type 1 diabetes, which is autoimmune, or Type 2 diabetes, which is linked to insulin resistance, Type 5 diabetes arises from nutritional deficiencies that impair the pancreas’s ability to function properly.
It most commonly affects lean young adults in low-income regions. For years, people with this condition were misdiagnosed as having Type 1 diabetes or an unusual form of Type 2, often leading to inappropriate treatments that failed to address the root cause.
Symptoms of type 5 diabetes can resemble those seen in other forms of diabetes, such as excessive thirst, frequent urination, persistent tiredness, unexplained weight loss, blurred vision, and slow-healing wounds. However, they are often paired with indications of malnutrition, including a lean physique, delayed growth or puberty in young people, anemia, and recurring infections.
These arise from nutritional deficiencies during early life rather than solely from insulin resistance or autoimmune causes, and they usually present before the age of 30, according to the International Diabetes Federation.
Researchers have been studying this condition for decades, particularly in parts of Asia and Africa. Yet, without official recognition, the disease remained poorly understood, and patients were rarely diagnosed correctly. Experts argued that grouping these patients under existing categories obscured the true cause of their illness.
The 2025 classification now formally separates Type 5 diabetes from other types, making it easier to study, identify, and manage. In India, where diabetes prevalence is already high, this recognition is particularly relevant. Tens of millions of people live with diabetes in the country, many of whom remain undiagnosed. Type 5 diabetes highlights a different pattern: it develops not from excess calories, but from too little nutrition during childhood, creating a double burden in regions where obesity and undernutrition coexist.
Although there is no new treatment specifically for Type 5 diabetes yet, official recognition marks a major step forward. Doctors can now approach lean patients with a history of malnutrition more carefully, avoiding a “one-size-fits-all” approach. Clearer classification could lead to better guidelines, more personalised care, fewer complications, and improved long-term outcomes.
Experts believe that understanding the role of childhood undernutrition in diabetes could eventually reshape how we prevent, monitor, and manage the condition—something countries like India urgently need as diabetes numbers continue to rise.
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An early sign of dementia can sometimes look like a common winter-related issue. When this symptom appears along with other warning signals, it may be wise to speak to a doctor. Dementia is a syndrome marked by a collection of related symptoms that point to a gradual decline in brain function. Over time, this can affect memory, behaviour, thinking, and even movement. In its early phase, however, dementia often shows up through subtle changes that are easy to overlook or mistake for something less serious.
Dementia UK notes that one possible early indicator of dementia is “low mood, anxiety or depression”. Its specialists explain: “In the early stages of dementia, people often begin to notice symptoms that interfere with day-to-day life.” The difficulty is that low mood or depression can also be linked to seasonal affective disorder (SAD), a form of depression that tends to appear during winter and ease as the days become longer and brighter.
The NHS lists the following possible symptoms of SAD:
The organisation explains: “A person experiencing early symptoms of dementia may notice these changes themselves, or they may be picked up first by family members, friends or colleagues. Memory problems are not always obvious in the early stages of some types of dementia, such as frontotemporal dementia, where changes in behaviour and personality may appear first.
“People with young onset dementia, where symptoms begin before the age of 65, are also less likely to have memory loss as an early symptom.”
The charity also points out that emotional changes can be linked to two specific types of dementia. In vascular dementia, a person may experience “changes in mood, behaviour and personality”, while Lewy body dementia can cause “mood changes, including anxiety and depression”.
That said, it is often difficult to know at first whether dementia is the cause of these warning signs. Dementia UK advises: “Many symptoms associated with dementia can also be caused by other physical or mental health conditions, such as thyroid disorders, menopause, vitamin B12 deficiency, depression, anxiety, work-related stress or relationship difficulties.
“This means that experiencing symptoms linked to dementia does not automatically mean someone has the condition. However, if you or someone close to you is showing signs or symptoms of dementia, it is important to visit a GP to understand what might be causing them.”
If you or someone you know is showing symptoms that resemble dementia, seeking advice from your GP is an important first step.
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