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Fasting may reset your body, but can it reset your mind? The new trend of dopamine fasting claims it can. And wait, there's more. This trend also works against dopamine resistance, implying that things that did not make you happier before will now do. You see, dopamine is a part of the brain's reward system and plays an important role in your pleasure reception. While this neurotransmitter is not directly linked to an individual's happiness, it triggers feelings of satisfaction, motivation and pleasure.
However, a person can also reach a stage of dopamine resistance if they continue to indulge in activities that trigger its frequent or constant release. In this case, the individual stops feeling the impact of this neurotransmitter and thus, does feel good or happy.
So does dopamine fasting work?
Dopamine fasting is a practice where individuals limit their exposure to activities or stimuli that typically provide a surge of dopamine. The idea behind dopamine fasting is to reset or recalibrate the brain's reward system. This is usually done by abstaining from gratifying things or experiences like social media, junk food, and even sex. Proponents of dopamine fasting argue that continual overstimulation from digital devices, social media, and easily accessible indulgences has numbed our brain's reward pathways. By regularly denying ourselves these dopamine triggers, the idea claims, we might restore our ability to acquire fulfilment from life's basic pleasures.
Having low levels of dopamine can make you less motivated and excited about things. In Parkinson's disease, there is not enough dopamine in the areas of the brain important for movement. This leads to problems with muscle stiffness and movements such as walking.
The symptoms of a dopamine imbalance depend on what is causing the problem. They include physical symptoms such as:
Adjusting dopamine levels is complicated, as it is involved in many different roles in the brain. Your doctor won't measure your dopamine levels directly, and there is no simple test to measure it. Your symptoms will be the clues that tell your doctor if you have too much or not enough dopamine. They will then prescribe medicines to adjust your dopamine level, based on your symptoms, and make adjustments based on how your body responds and how you feel.
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Most people reach for sunscreen when heading to the beach or stepping out for a long day in the sun. But skincare experts say sun protection should not be limited to vacations or outdoor activities. Sunscreen is meant to be part of a daily skincare routine.
This often raises a common question. If you are staying indoors all day, do you still need sunscreen?
Dermatologists say the answer depends on your surroundings and daily habits.
Many people assume that staying inside completely protects their skin from sunlight. But this is not always true.
Dr Khushboo Jha, MBBS, MD, Chief Dermatologist Consultant at Metro Hospital and Founder of One Skin Clinic, explains that sunlight entering through windows can still affect the skin.
“While standard window glass blocks most UVB rays, which cause sunburn, UVA rays can still pass through. These rays penetrate deeper into the skin and are linked to long term concerns such as premature aging, uneven pigmentation and loss of skin elasticity,” she says.
These UVA rays are often overlooked because they do not cause immediate redness or burning like UVB rays. However, over time they can lead to visible signs of skin aging and pigmentation.
Dermatologists say sunscreen indoors is particularly useful for people who spend long hours near windows or in well lit spaces.
Dr Jha notes that individuals who work near windows, sit in sunlit rooms or spend time driving during the day may still be exposed to sunlight. “Even short periods of daily exposure to sunlight over time can contribute to cumulative skin damage,” she explains.
In such situations, applying sunscreen in the morning can offer an added layer of protection. A broad spectrum sunscreen with at least SPF 30 is generally recommended.
This approach is especially relevant for people working in offices with large windows or those who frequently commute during daylight hours.
Experts also say sunscreen use indoors is not always equally necessary for everyone.
If you spend most of the day inside a room with minimal natural light and away from windows, your exposure to ultraviolet radiation becomes much lower.
Dr Jha says that in such cases the urgency of frequent sunscreen reapplication becomes less important. The risk of sun related skin damage is significantly reduced when there is little to no direct daylight entering the space.
This means sunscreen indoors should not be treated as a strict rule but rather as a flexible part of skincare based on lifestyle and environment.
Another topic that often comes up is blue light exposure from digital devices such as phones, laptops and tablets.
Some studies suggest that prolonged exposure to visible light may contribute to pigmentation, especially in individuals with deeper skin tones. However, dermatologists point out that the amount of blue light from electronic screens is much lower than what we receive from natural sunlight.
Dr Jha says the effect of digital screens on the skin is still being studied, but compared to sun exposure, the impact remains minimal.
Dermatologists suggest viewing sunscreen as a preventive skincare habit rather than a rigid rule.
Dr Jha recommends incorporating sunscreen into your morning routine, particularly if your day includes stepping outdoors or spending time in naturally lit environments.
In simple terms, if daylight reaches your workspace or you plan to go outside later in the day, applying sunscreen in the morning is a small step that can help protect your skin over time.
Credits: ABC News' Four Corners
At the age of 28, Courtney Paton realized she could never have children. She was medically infertile. This was after years of repeated surgeries, a total of seven, for 'suspected' endometriosis, due to which Dr Simon Gordon, Melbourne-based gynecologist removed both her ovaries and eventually her uterus.
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Her story came to light through an investigation by the Australian Broadcasting Company or ABC's Four Corners, an investigation that looked at the treatment she received from Dr Gordon.
Courtney says she trusted the doctor completely. Now she says that trust has been shattered. “I feel completely betrayed by not only Simon Gordon, but by Epworth, by the healthcare system,” she told the program.
Courtney first had laparoscopic surgery in 2018 with another surgeon, which confirmed she had endometriosis. The condition affects about one in seven Australian women and can cause severe pelvic pain and fertility problems.
Still struggling with pain, she began seeing Gordon in 2019 when she was 21.
Over the next several years she underwent seven surgeries with him. Gordon told her the procedures were necessary to treat severe endometriosis. Courtney and her family paid more than 32,000 Australian dollars for these surgeries alone.
But when investigators asked her to obtain her pathology reports, the results told a very different story. The tissue tests from most of her surgeries showed no evidence of endometriosis.
Despite this, operation reports written by Gordon continued to describe findings consistent with the disease.
Read: A Woman Lost Her Ovary To Endometriosis Surgery After Receiving An Ultimatum From Gynecologist
In 2021 Gordon removed one of Courtney’s ovaries, saying it was stuck to the pelvic wall. Later he removed the second ovary as well.
Independent specialists who reviewed the pathology for the investigation said the ovary appeared normal and there was no clear justification for removing it. One expert described the treatment as “unbelievable” after reviewing the medical records.
Medical guidelines generally advise caution when removing ovaries from young women who may want children in the future.
Despite losing both ovaries, Courtney continued to experience pelvic pain. Gordon later advised that she should undergo a hysterectomy.
Concerned, she sought a second opinion from another gynecologist who said the procedure was unnecessary and suggested non surgical treatments.
But after years of pain and repeated surgeries, Courtney says she felt desperate for relief and trusted the doctor who had treated her for so long. Her uterus was removed in 2023 when she was just 25.
Again, pathology results found no evidence of endometriosis.
Courtney is now pursuing legal action through a medical negligence claim. The case has also drawn attention from regulators, with investigations underway into Gordon’s conduct.
Australia’s federal health minister Mark Butler described the allegations as “physically sickening”.
For Courtney, the emotional impact remains overwhelming.
“No woman should ever have to endure what I’ve endured,” she said. “I’ve had the opportunity to have a family taken away from me.”
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Your DNA, or more specifically your genes, have fascinating interactions with your diet. These interactions are often bidirectional and form the basis of personalized nutrition through genomic biohacks.
This has impressive applications in solving some of the most stubborn health related challenges, including undesirable weight gain and obesity.
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Here are the five ways by which your DNA could positively shape your diet:
Nutrigenomics is the mechanism by which your diet affects your genes, and not the other way round. The process has massive implications for your health, and especially over how you can use specific dietary components to protect yourself against serious killer diseases like cancers and issues like faster aging.
Deficiency in key nutrients like Vitamin B9 or folate, Vitamin B12, choline & methionine can cause genomic instability and increased cancer risk.
In contrast, specific foods like curcumin, resveratrol, green tea, broccoli, Brussels sprouts etc can help with genomic stability and help fight inflammation, oxidative stress and cancers.
These are the processes by which your genes affect your diet or consumed food and hence central to our theme here. At times, nutrigenetics is referred to by the wider umbrella term nutrigenomics.
Your gene variants determine how you process specific nutrients, which explains why the same diet works differently for different people. Genetic tests like Eplimo can easily find this out.
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For instance, presence of certain variants of the SGK1 gene make those individuals more prone to high blood pressure from salt intake.
Similarly, variants in the FTO gene are strongly linked to obesity risk. Other very common examples are variants in the CYP1A2 gene that determine how fast you metabolize caffeine and mutations in the LCT gene that determines whether you are at risk of lactose intolerance.
Ghrelin, the hunger hormone, is produced mainly in the stomach and stimulates appetite, increases food intake as well as promotes fat storage.
The production of ghrelin hormone is governed by the GHRL gene, and a common variant in this gene called RS696217 is associated with unnecessary hunger and higher obesity risk.
Similarly, leptin is a hormone produced by the body’s fat tissues and regulates satiety or the feeling of fullness with regard to food.
Production of leptin is governed by the LEP gene, while its utilization is controlled by the LEPR (leptin receptor) gene. Variants in either, especially LEP, can cause severe, early-onset obesity.
Genes play a significant role in determining your metabolic pace. Studies show that genes account for up to 60 percent of the variations seen in the Resting Metabolic Rate between individuals.
RMR is basically a measure of how much calories are burnt while you are sitting or doing light activities.
It is different from Basal Metabolic Rate (BMR) that requires fasting and bed rest. Hundreds of genetic variants work together to set your RMR.
These include variants in the UCP1 gene governing thermogenesis or heat production, the MC4R gene that influences how the body burns nutrients for energy, and genes governing mitochondrial efficiency.
Building more muscles is a proven way to counter the negative impact of such variants and boost RMR.
Does your gut microbiome impact your genes more, or does your genes impact your gut microbes more? Definitely, it is the former, which is also a better known mechanism due to that greater impact.
But that doesn’t mean that the reverse impact, from your DNA to your microbiome which accounts for around 10 percent of its composition, is insignificant in any way.
For instance, your specific gene variants determine which bacteria thrive by influencing immune responses, metabolism, and food preferences.
Specific genes, such as the LCT gene, directly correlate with the abundance of beneficial bacteria like Bifidobacterium. Genetics also influence how you digest food and your dietary preferences, which in turn feeds specific bacterial species.
Other genetic factors too have been identified as having strong links to microbial diversity, which is a great marker for not only gut health, but overall health, performance and longevity.
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