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Fasting may reset your body, but can it reset your mind? The new trend of dopamine fasting claims it can. And wait, there's more. This trend also works against dopamine resistance, implying that things that did not make you happier before will now do. You see, dopamine is a part of the brain's reward system and plays an important role in your pleasure reception. While this neurotransmitter is not directly linked to an individual's happiness, it triggers feelings of satisfaction, motivation and pleasure.
However, a person can also reach a stage of dopamine resistance if they continue to indulge in activities that trigger its frequent or constant release. In this case, the individual stops feeling the impact of this neurotransmitter and thus, does feel good or happy.
So does dopamine fasting work?
Dopamine fasting is a practice where individuals limit their exposure to activities or stimuli that typically provide a surge of dopamine. The idea behind dopamine fasting is to reset or recalibrate the brain's reward system. This is usually done by abstaining from gratifying things or experiences like social media, junk food, and even sex. Proponents of dopamine fasting argue that continual overstimulation from digital devices, social media, and easily accessible indulgences has numbed our brain's reward pathways. By regularly denying ourselves these dopamine triggers, the idea claims, we might restore our ability to acquire fulfilment from life's basic pleasures.
Having low levels of dopamine can make you less motivated and excited about things. In Parkinson's disease, there is not enough dopamine in the areas of the brain important for movement. This leads to problems with muscle stiffness and movements such as walking.
The symptoms of a dopamine imbalance depend on what is causing the problem. They include physical symptoms such as:
Adjusting dopamine levels is complicated, as it is involved in many different roles in the brain. Your doctor won't measure your dopamine levels directly, and there is no simple test to measure it. Your symptoms will be the clues that tell your doctor if you have too much or not enough dopamine. They will then prescribe medicines to adjust your dopamine level, based on your symptoms, and make adjustments based on how your body responds and how you feel.
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Parkinson's disease is a progressive, neurodegenerative movement disorder caused by the loss of dopamine-producing brain cells, primarily affecting people over 60. Apart from motor loss, the disease also causes cognitive decline, depression, anxiety and swallowing problems.
The first symptom may be a barely noticeable tremor in just one hand or sometimes a foot or the jaw. Over time, swinging your arms may become difficult and your speech may become soft or slurred. The disorder also causes stiffness, slowing of movement and trouble with balance that raises the risk of falls.
However, before clear symptoms begin to appear, Neurologist Rachel Dolhun says certain signs may help identify the onset of the disease decades before it is diagnosed.
“It’s important to stress that not everyone who has these symptoms goes on to develop Parkinson’s,” said neurologist Rachel Dolhun. “But we know that in some people, these can be some of the earliest signs," she told The Washington Post.
Here is what you should look out for:
Loss of smell, or hyposmia, is a common and early non-motor symptom of Parkinson's disease, affecting up to 90 percent of patients. This symptom can significantly impact quality of life by reducing the enjoyment of food and diminishing appetite.
While strongly linked to Parkinson's, smell loss can also stem from other causes, including sinus problems, COVID-19, or aging.
Acting out dreams, known as REM Sleep Behavior Disorder (RBD), involves physically enacting vivid, often unpleasant dreams through shouting, punching, or kicking during sleep.
This typically happens because the brainstem fails to temporarily paralyze muscles during REM sleep. It is a strong early warning sign of Parkinson's disease, often appearing years or decades before motor symptoms. About 50 percent of people with Parkinson's experience RBD.
READ MORE: Parkinson’s Patients May Soon Walk Better With This New Personalized Brain Therapy
Constipation is a very common and significant non-motor symptom of Parkinson's disease that is caused by nerve changes slowing gut muscles and potentially exacerbated by low activity and dehydration.
Constipation can also be caused by Parkinson's medications such as anticholinergics, amantadine and other common drugs such as opioids, iron/calcium antacids.
The autonomic nervous system fails to properly constrict blood vessels or increase heart rate upon standing, often due to a lack of norepinephrine. This causes the autonomic nervous system to fail in regulating blood pressure. Over time, this leads to Neurogenic Orthostatic Hypotension.
Beyond dizziness, symptoms include blurred vision, weakness, fatigue, cognitive "fog," and "coat hanger pain" (pain in the neck/shoulders). Often times, patients experience dizziness in the morning or immediately after meals.
Diagnosing Parkinson’s disease is mostly a clinical process, meaning it relies heavily on a healthcare provider examining your symptoms, asking questions and reviewing your medical history. Various imaging and diagnostic tests used to detect disease includes CT scan, PET scan, MRI scan and genetic testing.
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Beyond icy roads and fogged-up car windscreens, the coldest season can also bring on a painful condition that leaves the skin covered in red, itchy patches. Doctors have issued a warning about this lesser-known illness, which tends to worsen in low temperatures.
The condition, known as cold urticaria, affects around one in 2,000 people. It causes swelling and itching of the skin when it comes into contact with cold air, cold water, or even air conditioning. Red welts or hives can appear within minutes, and the discomfort may last for as long as two hours.
Cold urticaria is an uncommon condition in which the body reacts abnormally to cold temperatures. It typically causes rashes or hives after exposure to cold air, water, food, or drinks. In some cases, symptoms can be more serious. According to the Cleveland Clinic, the condition may sometimes be linked to an underlying blood cancer or an infectious illness.
Griet Voet, head of a dermatology clinic in Ghent, Belgium, as per Express UK, said the condition is often confused with common winter skin problems such as eczema. “This is not just dry skin caused by cold weather, it is a genuine allergic reaction to cold,” she explained. In more severe cases, large areas of the body may be affected, particularly after swimming in cold water or spending extended periods outdoors. This can lead to intense itching, facial flushing, and even headaches, stomach pain, or fainting. Sudden temperature shifts, such as moving from a warm indoor space into cold outdoor air, can also trigger symptoms. Drinking ice-cold beverages may cause swelling of the lips, mouth, or throat.
Symptoms of cold urticaria differ from person to person and can range from mild to severe. They may be limited to a small patch of skin or spread across the entire body.
The most common sign is a skin rash that appears after contact with something cold. The rash usually develops once the exposure ends, as the skin begins to warm up.
The rash may include:
A medical professional can often diagnose cold urticaria using a simple test. An ice cube is placed on the skin, usually on the arm, for a few minutes and then removed. If a hive or rash appears several minutes later, the result is considered positive.
In cases of familial cold urticaria, diagnosis may involve exposure to cold air for a longer duration.
Doctors may also suggest blood tests to check for any underlying illness or infection that could be contributing to the condition.
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Health authorities have urged the public to stay alert to Nipah virus symptoms after doctors warned that up to 75 per cent of infected patients may not survive. The UK Health Security Agency (UKHSA) has classified Nipah as a “high priority pathogen” because of its severe fatality rate and the absence of any proven treatment.
In India, the federal health ministry has confirmed two cases in the eastern state of West Bengal. This has triggered large-scale containment measures, with local officials placing nearly 200 people who had contact with the infected individuals under quarantine.
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In response, several Asian nations have stepped up airport checks and health surveillance for travellers arriving from India. Professor Paul Hunter, an infectious disease specialist at the University of East Anglia, said identifying Nipah cases at borders is challenging, as symptoms can take a long time to appear.
According to UKHSA, Nipah virus is a zoonotic infection, meaning it can pass from animals to humans. It can also spread through contaminated food or via direct human-to-human contact. The virus was first discovered in 1999 during an outbreak affecting pig farmers in Malaysia and Singapore.
Fruit bats, especially those belonging to the Pteropus species, are the virus’s natural carriers. Research shows that Nipah can also infect other animals, such as pigs, dogs, cats, goats, horses and sheep.
Symptoms usually appear between four and 21 days after exposure, although longer incubation periods have occasionally been reported. More severe complications, including encephalitis or meningitis, can develop between three and 21 days after the initial illness begins.
Also Read: Nipah Virus Outbreak In India: All That You Need To Know About This Infection
UKHSA has cautioned that between 40 and 75 per cent of people infected with Nipah virus may die. Those who survive can experience long-term neurological effects, such as ongoing seizures or changes in behaviour and personality. In rare instances, the virus has been known to reactivate months or even years after the first infection.
For people travelling to regions where Nipah is known to occur, prevention largely involves reducing exposure risks:
Many Nipah infections have been linked to eating fruit or fruit-based products contaminated by the saliva, urine or droppings of infected fruit bats. Human-to-human transmission can also occur through close contact with an infected person or their bodily fluids, according to Mirror.
Such transmission has been documented in India and Bangladesh, with cases often involving family members or caregivers tending to infected patients. At present, there is no specific, proven treatment for Nipah virus infection, and no licensed vaccine is available to prevent it.
So far, no Nipah virus cases have been reported in the United States or the United Kingdom.
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