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We've all been there, you use the toilet, try to stand up, and suddenly your legs go numb. That odd pins-and-needles feeling can be surprising and uncomfortable. Though it might feel like a small inconvenience, it does have a scientific explanation. The numbness, also called transient paresthesia, happens when pressure blocks blood flow or presses on nerves in your lower extremities. It is normally harmless, but frequent occurrences can be signs of underlying health issues or poor toilet habits that must be addressed.
That weird numbness you experience after going to the bathroom is typically just a temporary annoyance, most often due to bad posture, straining, or sitting for an extended period. But if the numbness continues or gets worse, it is important to get medical guidance to make sure there are no underlying health issues. We discovered the top three reasons that could be responsible for this tingling and how can you avoid it? Let's dissect.
Struggling to push during a bowel movement can put excessive pressure on your abdomen and spine. This increased pressure can shift spinal discs, pressing against nerves that extend into your legs and feet. The result? A temporary loss of sensation, tingling, or weakness in your lower limbs.
Straining usually results from constipation, which in turn can be caused by a low-fiber diet, dehydration, or inactivity. If you notice that you're straining frequently, perhaps it's time to change your eating and drinking habits to help move your bowels more easily.
The way you sit on the toilet can also be a cause of that numbness in your legs. Most people are prone to hunching over when they are using their phones, reading, or just focusing too intensely. But this position can compress nerves and blood vessels in your pelvis, causing tingling or numbness.
When you sit slumped forward, you cut off blood supply to the lower half of your body, compressing nerves that travel from your pelvis to your toes. That's why the numbness will often radiate past your thighs and into your toes.
The more time you spend sitting on the toilet, the higher your chance of getting numb legs. Protracted sitting continually puts pressure on the nerves within your lower limbs, slowing blood flow and leaving you with the familiar pins-and-needles feeling.
If you habitually stay on the toilet for a long time, either from digestive problems or distractions such as browsing your phone, you may find that there is more numbness over the course of time. If constipation is leaving you on the toilet longer than normal, diet changes can calm your system.
Although periodic tingling is not a health issue, recurring numbness is a problem that needs to be addressed. Below are some professional-recommended ways of preventing it:
Being seated with your knees higher than your hips can make all the difference. Sitting this way enables your colon to unwind, facilitating smooth bowel movements while minimizing pressure on the lower parts of your body.
Don't slouch, as this squishes nerves and blood vessels, making numbness more likely. If necessary, lean your back against the toilet tank or wall to keep your posture good.
Specialists recommend five to ten minutes of toilet time per visit. If you are straining, stand up, walk around, and try later. Forcing the bowel movement can cause more damage than benefit, putting greater pressure on your spine and worsening numbness.
If constipation is a chronic problem, being hydrated and consuming fiber foods such as fruits, vegetables, and whole grains can get your digestive system back in working order.
Raising your feet using a toilet stool may position your body for a more natural and strain-free bowel movement. A squatting position keeps the rectal canal open, minimizing the need to push and reducing the risk of leg numbness.
Hard toilet seats can restrict circulation in your lower body, increasing the risk of numbness. A cushioned or padded toilet seat can provide better support, improving blood flow to the legs and feet while reducing pressure on the pelvis.
While it's normal to have some numbness in your legs from time to time when sitting on the toilet, ongoing tingling or numbness in your lower extremities may be a symptom of an underlying medical condition. If you find yourself experiencing:
It's best to see a healthcare expert to exclude conditions such as nerve compression, circulatory disorder, or spinal condition.
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An international team of researchers has developed an artificial intelligence (AI)-based tool that can significantly improve care for glaucoma -- a leading cause of irreversible blindness worldwide, according to a study published in The Lancet Primary Care journal today.
Researchers led by those from the University of Lisbon in Portugal found that the AI-based screening tool halved the number of unnecessary referrals for glaucoma.
The study, released during the Glaucoma Awareness Week, also showed an accuracy level at par with human eye doctors.
"The high accuracy at excluding people without glaucoma is especially important, as false alarms can lead to unnecessary hospital visits, patient anxiety, and added strain on healthcare services," the researchers said.
According to the researchers, AI-based screening could:
The study was carried out at a single screening center in Lisbon, Portugal, in 2023.
The experts screened 671 adults aged 55-65 for glaucoma via the AI tool, analyzing images of the eyes. The images were then independently graded by six glaucoma experts.
The AI-tool:
While modelling studies suggest that screening could substantially reduce glaucoma-related visual impairment and blindness, barriers include the need for specialised diagnostic equipment and trained personnel, particularly in low- and middle-income countries, and the intrinsically low positive predictive value of screening tests.
In such a scenario, the new study showed that "AI may provide a more viable option than population-wide screening", which may seem impractical.
Glaucoma is a chronic disease that affects an estimated 80 million individuals globally, according to the World Glaucoma Association.
It is a progressive, degenerative disorder of the optic nerve that produces characteristic visual field damage.
The disease stems from a long asymptomatic phase, resulting in substantial underdiagnosis and delayed treatment.
Even in high-income countries, up to 50 percent of individuals with glaucoma remain undiagnosed, frequently presenting moderate to advanced disease at first detection.
By the year 2040, it is estimated that there will be 22 million individuals worldwide who are blind from glaucoma.
When to see a doctor for glaucoma:
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In adults, long periods of sleep deprivation has been linked to problems such as weakened immunity, weight gain, depression, and an increased risk of dementia. However, scientists are now paying closer attention to how sleep affects the brain much earlier in life.
However, a new University of North Carolina School of Medicine study suggests that disrupted sleep during early childhood may interfere with key stages of brain development and asl well as increase the risk of developing autism.
Sleep plays a crucial role in helping these synapses form and strengthen. During sleep, the brain organizes and stabilizes these neural connections, shaping the foundation for future brain function. If sleep is repeatedly disrupted during this delicate stage of development, the process may be affected.
Frequent waking or sleep disturbances could interfere with how these neural connections are formed, potentially influencing behavior and cognitive abilities later in life.
“The unique effects of sleep loss during development are largely unexplored,” Diering said. “Our data show that babies and children are more vulnerable to the negative effects of sleep disruption. We also found that sleep loss during this crucial period of time can negatively interact with underlying genetic risk for autism spectrum disorder.”
Sleep problems are already known to be common in people with autism. In fact, sleep disruption has been reported in more than 80 percent of individuals with autism spectrum disorder. However, researchers have long debated whether these sleep issues are a cause of the disorder or a consequence of it. Understanding how sleep interacts with brain development could help scientists detect autism earlier and potentially develop new treatment strategies.
In earlier work conducted in 2022, researchers examined how sleep disruption during early life might interact with genetic factors linked to autism. Using mouse models, they disrupted sleep during the third week of life, a developmental stage roughly comparable to ages one to two in humans.
The study found that sleep disruption during this period produced long lasting behavioral changes. Male mice that were genetically vulnerable to autism showed deficits in social behavior later in life. These results suggested that sleep disruption during critical stages of development may interact with genetic risk factors in ways that shape long term behavior.
To investigate further, researchers studied how developing and adult mice respond differently to sleep deprivation.
Using specially designed housing systems equipped with sensitive sensors, scientists tracked the animals’ breathing and movement. This allowed them to determine when the mice were awake and when they were asleep.
The researchers observed that adult mice were able to compensate for lost sleep. After experiencing sleep deprivation, the adults increased their sleep later during their normal active period. This process, known as sleep rebound, allowed them to recover some of the lost rest.
Younger mice behaved very differently. They showed no sleep rebound at all, meaning they did not compensate for the sleep they had lost. This finding suggests that younger brains may be far more vulnerable to the effects of sleep disruption.
The consequences were also visible in cognitive performance. Sleep deprived young mice performed poorly on learning and memory tasks, while adult mice were significantly more resilient after losing sleep.
The results showed that sleep deprivation in young mice significantly altered the formation of synapses. These changes were not seen in adult mice. “This now provides one of the largest and most comprehensive datasets to examine the molecular effects of sleep loss across the lifespan,” Diering said.
“Development is not something that one can go back and do again,” Diering said. “Sleep is important for the entire life and especially during development. Understanding what we know now will place greater emphasis on understanding sleep issues in ASD and could lead to an important therapeutic avenue to treat ASD and other developmental conditions.”
The findings highlight an important message for parents and caregivers. During early childhood, healthy sleep patterns may play a critical role in shaping the brain for years to come.
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Using amphetamines, cocaine and cannabis can significantly increase your risk of having a brain stroke particularly among younger adults, according to a new University of Cambridge analysis
The findings highlight how recreational drug use may contribute to a preventable health risk, especially among people under the age of 55.
Megan Ritson, a stroke genetics researcher at the University of Cambridge and lead author said the results provide strong evidence linking certain drugs to stroke risk.
“These findings provide compelling evidence that drugs like cocaine, amphetamines, and cannabis are causal risk factors for stroke,” Ritson noted.
A stroke occurs when blood flow to part of the brain is interrupted. This can happen when a blood vessel becomes blocked by a clot, known as an ischemic stroke, or when a blood vessel bursts and causes bleeding in the brain, known as a hemorrhagic stroke. Both types can lead to serious brain damage and can be life threatening.
When researchers combined data from eight previous studies, they found that recreational amphetamine use was associated with more than double the risk of stroke across all adult age groups. For individuals under the age of 55, the increase was even greater. In this group, amphetamine use was linked to nearly triple the risk of stroke compared with people who do not use the drug.
Across all age groups, the analysis found that amphetamine use increased the risk of ischemic stroke by 137 percent and hemorrhagic stroke by 183 percent. These figures reflect relative risk, meaning the probability of stroke is higher among users compared with non users.
The analysis showed that cocaine use nearly doubled the risk of stroke of any kind and more than doubled the risk of hemorrhagic stroke. Additional genetic investigations were conducted alongside the main analysis to better understand whether the relationship might be causal rather than simply linked to other lifestyle factors.
Eric Harshfield, a genetic epidemiologist at the University of Cambridge, said the findings suggest the drugs themselves may play a direct role. “Our analysis suggests that it is these drugs themselves that increase the risk of stroke, not just other lifestyle factors among users,” Harshfield said.
Among people under the age of 55, cannabis use was linked to a 14 percent increase in stroke risk. Although the increase is lower than that associated with stimulant drugs, researchers say it remains important because cannabis is widely used.
The researchers also note that many of the studies included in the analysis relied on participants reporting their own drug use. Because of this, other lifestyle factors could potentially influence the results. Further research will be needed to better understand the biological mechanisms involved and how different patterns of drug use may affect stroke risk. Still, scientists say the evidence now available provides an important foundation for future public health strategies.
“These findings give us stronger evidence to guide future research and public health strategies,” Ritson said.
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