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Some days my brain is like a storm, thoughts moving faster than I can keep up. A small mistake becomes an catastrophe, an offhand remark becomes a soul-deep fear. I turn around and around, analyzing each word, every move, every potentiality. But then, I discovered recently this easy 20-second hack which was actually pretty straightforward but made a tremendous difference in the negative thinking. Quickly [sitting my hand on my heart and reminding myself, I am enough. Even just that small hesitation interrupts the madness. My breath slows, my shoulders ease, and for a moment, the hurricane calms. This practice over time has become my anchor, reminding me that I am not thoughts—I am so much more.
Researchers at the University of California, Berkeley, have discovered that it doesn't need to take long to practice self-compassion to be beneficial. The study, published in the Behaviour Research and Therapy journal, revealed that performing a 20-second self-compassion touch, such as putting a hand on your heart or belly, can greatly reduce levels of stress and anxiety.
According to psychology researcher Eli Susman, who co-authored the study, a group of 135 college students was asked to dedicate just 20 seconds a day to affirm themselves with kind and positive thoughts while engaging in a self-compassionate touch. The results were striking: those who consistently practiced this simple technique over a month experienced notable improvements in mood, self-compassion, and emotional resilience, while stress hormone levels decreased.
Why 20 Seconds of Self-Compassion Works
1. Decrease in Cortisol Levels
The stress hormone cortisol is the cause of much of the physical and emotional damage chronic stress inflicts on the body. The researchers discovered that a mere 20 seconds of self-compassionate touch resulted in a measurable drop in cortisol, allowing people to recover from stress more rapidly.
2. Better Emotional Well-Being
By practicing positive self-affirmation and empathetic touch, study participants reported greater emotional equanimity and reduced reactivity to stressful challenges.
3. A Simple, Accessible Practice
Unlike many conventional mindfulness practices that might demand lengthy meditation sessions, this micropractice requires only 20 seconds, rendering it simple to fit into daily activities, be it at home, the workplace, or even during public transport rides.
How to Practice Self-Compassionate Touch
This exercise is very easy and can be done anywhere. Here's how you can adapt it to your daily life:
Step 1: Recognize Your Emotions
Close your eyes and reflect on a recent experience that made you feel stressed, unworthy, or critical of yourself. Notice the sensations in your body as you reflect on this episode.
Step 2: Practice a Soothing Touch
Put one hand on your heart and the other on your belly. If this doesn't feel comfortable to you, you can experiment with other ways of self-compassionate touching, including:
Stroking the back of your neck
Rubbing a place on your palm with your thumb
Hugging yourself lightly by holding your arms in across your chest
Step 3: Breathe Deeply and Give Yourself Kindness
Take a slow, deep breath in. Feel the warmth and gentle pressure of your hands. As you exhale, focus on releasing tension. Now, in your mind, repeat self-compassionate affirmations such as:
“I am kind to myself.”
“I am not my mistakes.”
“I give myself room and comfort.”
“I celebrate my uniqueness.”
“I take this time to appreciate who I am.”
Step 4: Finish with a Sense of Gratitude
Open your eyes after 20 seconds and simply take a moment to admire yourself for taking the time to do this practice. You can repeat it as many times as you need throughout the day.
Susman calls this approach a "micropractice"—a tiny but effective habit that enhances mental health without taking up much time. These practices are based on classic mindfulness and meditation practices but are tailored to fit today's busy lives.
While the research was conducted with college students, the findings have applications for individuals of all ages. Whether you are a working professional with a packed schedule, a parent with numerous responsibilities, or an individual dealing with anxiety, adding a 20-second self-compassion exercise to your daily routine can be a convenient and effective method for managing stress and developing resilience.
Making It a Daily Habit
The secret to reaping the rewards of self-compassionate touch is consistency. Below are some ways to incorporate it into your daily life:
Begin your day by practicing self-compassion in bed before rising.
Utilize it as a fast tool during stressful situations at work or school.
Unwind by doing this micropractice before bedtime to relax.
May merely 20 seconds a day cause you to desist from spinning? The short answer, per the most up-to-date science, is that yes, it can. Micropractices for self-compassion provide a straightforward, research-supported means for lessening distress, enhancing emotional resilience, and cultivating a friendlier relationship with oneself.
In a world where stress and worry are escalating, this simple practice is a good reminder that simple, purposeful acts of care for ourselves have the ability to create tremendous transformations in our mindset. Why not give it a try for one month, you might find a surprising transformation.
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Malaria infected an estimated 282 million people and caused about 6,10,000 deaths worldwide in 2024, according to the World Health Organization’s (WHO) latest World Malaria Report. The report placed strong emphasis on drug resistance, warning that it remains one of the biggest threats to global elimination goals.
WHO-recommended vaccines helped prevent roughly 170 million cases and one million deaths last year, which is about nine million more than the year before. Around 95 per cent of malaria deaths occurred in the African Region, with children under five forming the largest share.
Within the WHO South-East Asia Region, India represented 73.3 per cent of all malaria cases and 88.7 per cent of all malaria-related deaths. The report also underscored that the world is nowhere close to meeting the targets set under the Global Technical Strategy for malaria 2016–2030.
However, a group of University of Nottingham researchers have now found a key protein that is an enticing target for new antimalarial interventions. The study looked at a protein called Aurora-related kinase 1 (ARK1), which plays an important role in the parasite’s unusual cell division.
ARK1 helps control the parasite’s mitosis (cell division) and organizes a structure called the spindle, which separates genetic material so new parasites can form.
Scientists turned off the ARK1 gene using genetic engineering techniques to see what would happen. Without ARK1, the parasites could not form proper spindles and failed to reproduce, suggesting the protein could be a weak spot that future malaria treatment.
"What makes this discovery so exciting is that the malaria parasite's 'Aurora' complex is very different from the version found in human cells," senior author Rita Tewari said.
Anopheles stephensi is a malaria-transmitting mosquito originally found in South Asia. Unlike many other malaria vectors, it thrives in cities and breeds in man-made water sources such as storage tanks, containers, and discarded tyres. It can carry both Plasmodium falciparum and P. vivax parasites.
In recent years, this mosquito has spread into several African countries, where it adapts easily and shows resistance to multiple insecticides. This expansion has increased the threat of urban malaria outbreaks, as highlighted by the World Health Organization.
At present, Anopheles stephensi has been detected in nine African countries and is proving difficult to control due to widespread insecticide resistance.
The report noted that WHO approved the world’s first malaria vaccines in 2021, and 24 countries have now added them to their regular immunisation schedules. Dr Tedros Adhanom Ghebreyesus, WHO Director-General, said that new preventive tools provide reason for optimism, but many obstacles remain.
He pointed out the rise in cases and deaths, the pressure from drug resistance, and the impact of reduced funding. These factors could undermine the progress achieved over the last twenty years.
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Men planning to conceive should practise caution while consuming antioxidant supplements, as excessive intake may affect sperm and early craniofacial development in babies, a Frontiers in Cell and Developmental Biology study shows.
Antioxidants are often promoted as powerful supplements that can help protect the body from chronic conditions, including cancer, chronic obstructive pulmonary disease and dementia.
However, researchers at the Texas A&M College of Veterinary Medicine and Biomedical Sciences (VMBS) found that consuming high levels of the antioxidant compounds N-acetyl-L-cysteine (NAC) and selenium (Se) can alter skull and facial shape in female children, even though the treated fathers did not exhibit obvious health problems.
Dr Michael Golding, a professor in the VMBS’ Department of Veterinary Physiology and Pharmacology, whose research team made the discovery said: "We know alcohol causes oxidative stress and we were looking to push back on it by adding a supplement known to lower oxidative stress.
"When we realized that offspring born to males that had only been given NAC were displaying skull and facial differences, it was a surprise because this molecule is universally thought to be good.
“When we sat down to think it through, we realized that it makes sense — you take a multivitamin to ensure that you’re in balance, but if the thing that you’re taking to ensure you’re in balance is unbalanced (the dose of antioxidants is too high), then you’re not doing a good thing.
“Sperm health is another performance metric; it’s just not one that we think about in everyday life. If you’re taking a high-dose antioxidant, you could be diminishing your reproductive fitness, and part of the journey toward the bad outcome is going to be the effects on the offspring.”
Antioxidants are important because they protect the fundamental structure of the human body, cells. They safeguard an individual's DNA and proteins from the damage caused by free radicals.
Research has shown that a diet high in antioxidants may help individuals prevent chronic diseases and mental health issues. But on the contrary, having too many antioxidants in your body could confuse your cells' responses, leading to more damage than intented.
Antioxidants basically neutralize free radicals in the body. These free radicals are extra atoms that are produced during the body's internal processes and also by some external factors.
Overproduction of these free radicals, during the process of oxidation in the human body, damages cell membranes and other structures, including cellular proteins, lipids and DNA. Oxidation can be exacerbated by stress, smoking, alcohol, sunlight and pollution.
In the long run, they can lead to diseases such as cancer and heart diseases. It is pertinent to note that the brain is prone to oxidative stress due to its high metabolic activity. Here, the role of antioxidants becomes particularly important as they fight oxidative stress which could otherwise lead to stroke, traumatic brain injury or neurodegenerative diseases like Alzheimer's.
If you take large amounts of antioxidants, then it could hamper the cell's defence mechanisms and normal signalling. Different types of antioxidants also have different properties, so they may not be interchangeable. Therefore, health experts advise people to be mindful of the amount of antioxidant-rich food they are including in your diet. Notably, studies have found that antioxidant supplements have a lower impact such as natural food items.
Here Are Top 10 Antioxidant Rich Food:
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A simple biomarker blood test can now detect dementia in women 25 years before symptoms appear, a JAMA study suggests.
Researchers from the University of California San Diego have found a protein in the blood called plasma phosphorylated tau 217 (p-tau217), a protein linked to the brain changes seen in Alzheimer’s disease.
The researchers found a strong association between higher levels of p-tau217 in the blood and a higher chance of developing both mild cognitive impairment (MCI) and dementia, a key contributer in the developemnt of Alzheimer’s.
Neuroscientist Linda McEvoy, from the Kaiser Permanente Washington Health Research Institute: "Blood-based biomarkers like p-tau217 are especially promising because they are far less invasive and potentially more accessible than brain imaging or spinal fluid tests.
"This is important for accelerating research into the factors that affect risk of dementia and for evaluating strategies that may reduce risk."
The connection between higher p-tau217 and dementia was stronger in women over 70, the researchers also found, and in those carrying the APOE ε4 gene that has been linked to Alzheimer's disease in the past.
Dementia is an umbrella term used to describe a significant decline in mental function that is serious enough to affect everyday life. It commonly impacts memory, thinking, and reasoning skills.
Dementia itself is not a single disease but a collection of symptoms caused by underlying conditions such as Alzheimer’s disease or vascular dementia.
Common signs include memory problems, confusion, difficulty finding words, changes in mood or behaviour and trouble completing familiar tasks.
These symptoms usually worsen over time and are not considered a normal part of ageing. Although there is no cure, treatment options can help manage symptoms, and early diagnosis plays an important role in care planning.
Alzheimer's disease is one of the most common forms of dementia and mostly affects adults over the age of 65.
About 8.8 million Indians aged 60 and above are estimated to be living with Alzheimer's disease. Over seven million people in the US 65 and older live with the condition and over 100,00 die from it annually.
Alzheimer's disease is believed to be caused by the development of toxic amyloid and beta proteins in the brain, which can accumulate in the brain and damage cells responsible for memory.
Amyloid protein molecules stick together in brain cells, forming clumps called plaques. At the same time, tau proteins twist together in fiber-like strands called tangles. The plaques and tangles block the brain's neurons from sending electrical and chemical signals back and forth.
Over time, this disruption causes permanent damage in the brain that leads to Alzheimer's disease and dementia, causing patients to lose their ability to speak, care for themselves or even respond to the world around them.
While there is no clear cause of Alzheimer's disease, experts believe it can develop due to genetic mutations and lifestyle choices, such as physical inactivity, unhealthy diet and social isolation.
Early symptoms of Alzheimer's disease include forgetting recent events or conversations. Over time, Alzheimer's disease leads to serious memory loss and affects a person's ability to do everyday tasks.
There is no cure for this progressive brain disorder and in advanced stages, loss of brain function can cause dehydration, poor nutrition or infection. These complications can result in death.
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