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Some days my brain is like a storm, thoughts moving faster than I can keep up. A small mistake becomes an catastrophe, an offhand remark becomes a soul-deep fear. I turn around and around, analyzing each word, every move, every potentiality. But then, I discovered recently this easy 20-second hack which was actually pretty straightforward but made a tremendous difference in the negative thinking. Quickly [sitting my hand on my heart and reminding myself, I am enough. Even just that small hesitation interrupts the madness. My breath slows, my shoulders ease, and for a moment, the hurricane calms. This practice over time has become my anchor, reminding me that I am not thoughts—I am so much more.
Researchers at the University of California, Berkeley, have discovered that it doesn't need to take long to practice self-compassion to be beneficial. The study, published in the Behaviour Research and Therapy journal, revealed that performing a 20-second self-compassion touch, such as putting a hand on your heart or belly, can greatly reduce levels of stress and anxiety.
According to psychology researcher Eli Susman, who co-authored the study, a group of 135 college students was asked to dedicate just 20 seconds a day to affirm themselves with kind and positive thoughts while engaging in a self-compassionate touch. The results were striking: those who consistently practiced this simple technique over a month experienced notable improvements in mood, self-compassion, and emotional resilience, while stress hormone levels decreased.
Why 20 Seconds of Self-Compassion Works
1. Decrease in Cortisol Levels
The stress hormone cortisol is the cause of much of the physical and emotional damage chronic stress inflicts on the body. The researchers discovered that a mere 20 seconds of self-compassionate touch resulted in a measurable drop in cortisol, allowing people to recover from stress more rapidly.
2. Better Emotional Well-Being
By practicing positive self-affirmation and empathetic touch, study participants reported greater emotional equanimity and reduced reactivity to stressful challenges.
3. A Simple, Accessible Practice
Unlike many conventional mindfulness practices that might demand lengthy meditation sessions, this micropractice requires only 20 seconds, rendering it simple to fit into daily activities, be it at home, the workplace, or even during public transport rides.
How to Practice Self-Compassionate Touch
This exercise is very easy and can be done anywhere. Here's how you can adapt it to your daily life:
Step 1: Recognize Your Emotions
Close your eyes and reflect on a recent experience that made you feel stressed, unworthy, or critical of yourself. Notice the sensations in your body as you reflect on this episode.
Step 2: Practice a Soothing Touch
Put one hand on your heart and the other on your belly. If this doesn't feel comfortable to you, you can experiment with other ways of self-compassionate touching, including:
Stroking the back of your neck
Rubbing a place on your palm with your thumb
Hugging yourself lightly by holding your arms in across your chest
Step 3: Breathe Deeply and Give Yourself Kindness
Take a slow, deep breath in. Feel the warmth and gentle pressure of your hands. As you exhale, focus on releasing tension. Now, in your mind, repeat self-compassionate affirmations such as:
“I am kind to myself.”
“I am not my mistakes.”
“I give myself room and comfort.”
“I celebrate my uniqueness.”
“I take this time to appreciate who I am.”
Step 4: Finish with a Sense of Gratitude
Open your eyes after 20 seconds and simply take a moment to admire yourself for taking the time to do this practice. You can repeat it as many times as you need throughout the day.
Susman calls this approach a "micropractice"—a tiny but effective habit that enhances mental health without taking up much time. These practices are based on classic mindfulness and meditation practices but are tailored to fit today's busy lives.
While the research was conducted with college students, the findings have applications for individuals of all ages. Whether you are a working professional with a packed schedule, a parent with numerous responsibilities, or an individual dealing with anxiety, adding a 20-second self-compassion exercise to your daily routine can be a convenient and effective method for managing stress and developing resilience.
Making It a Daily Habit
The secret to reaping the rewards of self-compassionate touch is consistency. Below are some ways to incorporate it into your daily life:
Begin your day by practicing self-compassion in bed before rising.
Utilize it as a fast tool during stressful situations at work or school.
Unwind by doing this micropractice before bedtime to relax.
May merely 20 seconds a day cause you to desist from spinning? The short answer, per the most up-to-date science, is that yes, it can. Micropractices for self-compassion provide a straightforward, research-supported means for lessening distress, enhancing emotional resilience, and cultivating a friendlier relationship with oneself.
In a world where stress and worry are escalating, this simple practice is a good reminder that simple, purposeful acts of care for ourselves have the ability to create tremendous transformations in our mindset. Why not give it a try for one month, you might find a surprising transformation.
Credit: Bryan Johnson/X
Bryan Johnson, the billionaire biohacker and longevity enthusiast, has been diagnosed with an incurable autoimmune disease called Autoimmune Gastritis (AIG).
Johnson is known for his radical longevity experiments, including measuring the biological age of his organs in an effort to reverse aging, injecting himself with ketamine, taking over 50 pills a day, undergoing fat transfers, and receiving blood transfusions from his teenage son.
In a recent post on X, he made the shocking announcement of his autoimmune conditions, which went undetected for years: Autoimmune Gastritis.
Calling it "bad news," Johnson wrote: "I have an autoimmune disease. My stomach is eating itself."
"2-5 per cent of people have this, too. Likely more, because it hides," he added.
According to Johnson, his stomach had been attacking itself without causing noticeable symptoms. The condition was only discovered in May.
Autoimmune Gastritis, a condition in which the immune system attacks the stomach's acid-producing cells, also impairs the absorption of iron and vitamin B12.
Johnson said one of the earliest signs of the condition was persistently low ferritin levels over the past 11 years despite not having anemia.
"We continually tried to raise my iron levels with food and supplementation, but nothing would work."
He said he followed a plant-based diet, trained intensely, used a sauna and hyperbaric oxygen therapy, and took iron supplements, yet his iron levels remained low.
Ferritin stores the body's iron. Iron is essential for transporting oxygen and producing energy, and low iron levels can lead to fatigue, weakness, and dizziness.
Johnson said autoimmune gastritis is difficult to diagnose because its earliest clue is often overlooked.
"The earliest clue, low ferritin, is the one standard medicine waves through. Low iron stores get normalized and rarely investigated at all when anemia hasn't shown up yet. That blind spot is what hid mine for a decade."
While initially it wasn't clear why his iron levels were continuously dipping, after further testing, Johnson's team identified three interconnected issues.
Autoimmune Gastritis was preventing normal iron absorption by damaging the stomach cells that produce acid. He also discovered autoimmune thyroid disease.
"The iron deficiency, the autoimmune gastritis driving it, and the autoimmune thyroid disease alongside it. Iron and thyroid feed each other both ways; low iron impairs the conversion of thyroid hormone into its active form, and an underactive thyroid impairs how the body uses iron," Johnson explained.
According to research published in JAMA Network Open, about one in four Americans may have inadequate iron intake or absorption.
As per the study of more than 8,000 Americans, 14 per cent of adults had absolute iron deficiency, reflecting depleted iron stores.
Even after excluding common causes such as anemia, pregnancy, heart failure, and chronic kidney disease, 11 per cent remained iron deficient. Another 15 per cent had functional iron deficiency, where iron levels appear normal but the body cannot use the mineral effectively.
Since receiving the diagnosis, Johnson has begun iron infusions, which deliver a full dose of iron in a single treatment.
He said his team will continue routine monitoring of his ferritin and iron levels.
Johnson ended his post with a warning that hidden health conditions can go unnoticed for years.
"You too may have a lurking health issue that is undiagnosed and could increase in severity from unhealthy life choices, without your knowing… A gentle nudge that minding your health, no matter your situation in life, is a good decision-making."
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A recent study has indicated that following a healthy diet consistently may help reduce the risk of dementia, even in those who already show early signs of Alzheimer's disease. The study could help in early management of neurodegenerative diseases like dementia.
According to a new study published in the JAMA Network Open, scientists found that participants who maintained better-quality diets over a period of 15 years were less likely to develop dementia, regardless of whether their blood tests indicated the presence of Alzheimer ’s related biomarkers.
The findings also suggest that healthy eating habits may benefit even after the individual shows signs of the disease processes have begun.
The researchers followed middle-aged and older adults one and a half decades, examining their dietary habits alongside blood biomarkers associated with Alzheimer's disease, including phosphorylated tau217 (p-tau217), glial fibrillary acidic protein (GFAP), and neurofilament light (NfL).
These biomarkers are commonly used to detect early Alzheimer's-related changes and neurodegeneration before symptoms of the disease become visible.
The participants followed a healthy diet, particularly a Mediterranean-style as well as an anti-inflammatory diet. The findings showed that individuals with healthier diets had a lower risk of developing dementia over the follow-up period.
Remarkably, this association was observed even among participants who presented Alzheimer's biomarkers, indicating that diet may offer protective benefits despite early signs.
Alzheimer's disease begins years, and sometimes decades, before memory problems emerge. During this preclinical stage, abnormal proteins accumulate in the brain while individuals remain symptom-free.
Read more: New Blood Test May Predict Alzheimer's Symptoms At Least 4 Years In Advance: Study
The new findings indicate that lifestyle factors such as diet could still influence dementia risk even after these biological changes have started. This reinforces growing evidence that preventive measures need not be limited to people without detectable Alzheimer's pathology.
While the study shows promising results of the effect of a healthy diet on the neurodegenerative disease, it does not prove that diets can directly prevent dementia.
The researchers emphasized that the study was merely an observation, meaning it highlighted an association rather than a direct cause-and-effect relationship. It just supports the idea that long-term healthy eating could have a significant impact on one’s brain health throughout adulthood.
The anti-inflammatory diet, as recommended by Harvard Nutrition Source, helps calm the immune system by choosing foods that reduce inflammation.
This dietary approach encourages a balance of fruits, vegetables, healthy fats, whole grains, and other nutrient-dense foods while avoiding those that trigger inflammation.
On the other hand, Mediterranean diet, long celebrated for its heart-and longevity-promoting benefits, is now gaining attention for its beneficial effects on the brain.
This diet emphasizes plant-based foods, healthy fats such as extra virgin olive oil, nuts, fruits, vegetables, and whole grains, with moderate fish intake.
In another study published in Nature Medicine, researchers observed that individuals at the highest genetic risk for Alzheimer’s disease, particularly carriers of the APOE4 gene variant, showed the most significant reduction in dementia risk when adhering closely to this dietary pattern.
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Parkinson’s disease is the second most common neurodegenerative disorder in the world and is the fastest-growing neurological condition worldwide. Its classic hallmarks - resting tremor, bradykinesia, and rigidity usually lead to diagnosis only after extensive dopaminergic neuronal loss has already happened. Newer research, however, highlights a prodromal window that might open decades earlier. There’s an increasing theory that pathological changes of the nervous system could start in a person’s 20s. This means that early detection is not just desirable but could be transformative for clinical outcomes.
Individuals with PD at prodromal and early motor stages alike report symptoms in multiple domains, including behavioral, cognitive, autonomic, sensory, sleep-related, and activities of daily living. Most of these symptoms are subtle and overlap with common conditions, so they aren’t often recognized or are mistaken for ageing, stress or other lifestyle factors. So, identifying consistent patterns amid daily behavioral variations is crucial for improving early PD detection.
Sleep Disturbances: An Early Warning Sign
One of the most robust prodromal markers is REM sleep behavior disorder (RBD). People who are affected have sleep disruption, physically act out their dreams during REM phase (acting out dreams), and vivid dream life and some through sleepwalking — all of them worthy substrates for signals of early brainstem pathology.
Anosmia, a partial or complete loss of the sense of smell, could be PD’s earliest recognizable sign, occurring as much as 10 years before motor signs become apparent. In practice, this means being unable to easily perceive familiar smells like food or coffee. Because this symptom manifests so early and appears so unrelated to the health of the brain, it is rarely taken seriously in clinical practice.
Chronic constipation is a common prodromal symptom indicating reduced gut motility that can predate motor symptoms by years. This observation is consistent with the gut–brain axis hypothesis: gut microbiota dysbiosis disrupts gastrointestinal motility, permeability and inflammation, which may facilitate a prion-like transmission of misformed alpha-synuclein (α-syn) from gut to brain through the enteric nervous system.1 further underscoring the biomarker potential of gastrointestinal symptoms with clinical relevance.
Before tremors are apparent, there can be subtle changes in fine motor control. Trouble with tasks such as handwriting, using electronic devices or manipulating small objects, along with uncharacteristic anxiety or a low mood, can serve as signals of the preclinical stage. Micrographia (progressively smaller and cramped handwriting) is a particularly telling sign from daily life that deserves a neurological workup when it appears without an obvious cause.
Fatigue that never seems to get better with rest affects work performance, social engagement and daily motivation, and can occur long before an official diagnosis. More than half of all PD patients develop at least one form of autonomic dysfunction, which can precede motor symptoms by four years or more, and is now being recognized as a key prognostic biomarker for prodromal PD. Cardiovascular instability, orthostatic hypotension, and urinary irregularities further influence how individuals navigate everyday environments long before a definitive diagnosis is made.
The evidence reviewed here collectively supports a paradigm shift: Parkinson's disease is best defined as systemic, progressive, and with recognizable signals in daily life long before motor signs of decline. Disrupted sleep, anosmia, gastrointestinal changes, fine motor difficulties and chronic fatigue are not complaints in a vacuum; they are potential early signs of a neurological process left unsupervised and now in motion. Incorporating routine clinical assessment of these behavioural precursors and pre-motor signs would allow us to meaningfully extend the opportunity for therapeutic intervention, which could in turn improve patient outcomes across a broad range of CNS disorders.
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