Credits: Canva
The world that we live in is filled with sounds, some are comforting and some could be jarring too. But what if the constant noise surrounding us is doing us more harm than we realize? There has been immense research that shows that noise is not just a nuisance, but a silent killer and affects our health in ways we do not even know. There are associations of sound causing heart attacks, type 2 diabetes, and dementia.
Noise is seen as an annoyance, but it effects can go beyond what we imagine. When we hear a sound, it travels through the ear to the brain. This is where it gets processed by the amygdala. It is a region that is responsible for emotional responses. This also triggers a stress response- our heart rate increases, our blood pressure rises, and stress hormones like cortisol flood our system.
This response is also designed to help us react to immediate threats. Especially, if we hear the sound of a predator approaching. However, when we are exposed to constant noise, this response is triggered repeatedly and could compel us to live in a long-term anxious state.
Many studies including Harvard Health and theAmerican College of Cardiology have found associations of noise with health problems including putting a person at a higher risk of cardiovascular diseases such as heart strokes, attacks, and high blood pressure. The constant activation of the stress response can take a toll on the body, increasing inflammation and making it harder for the heart and circulatory system to function properly. Over time, this can lead to serious health conditions like heart disease and diabetes.
Even more troubling, research suggests that noise pollution may contribute to mental health issues. Studies have found a strong connection between exposure to noise and disturbed sleep, which in turn can cause anxiety, depression, and cognitive decline. The World Health Organization estimates that noise contributes to around 12,000 premature deaths annually across Europe alone. This invisible threat, however, is often overlooked because the effects are gradual and cumulative.
One of the most insidious aspects of noise pollution is its impact on sleep. Even when we are asleep, our bodies are not fully immune to the effects of sound. Our ears never fully “turn off,” meaning that even faint noises can disrupt our sleep cycle. Research has shown that people who live in noisy environments—whether near busy roads, airports, or urban centers—often experience fragmented sleep, leading to fatigue and a weakened immune system. Over time, this chronic lack of restful sleep can lead to significant health problems, including an increased risk of developing cognitive disorders such as dementia.
As cities continue to grow, noise pollution is becoming more widespread. Traffic noise, in particular, is one of the most common and harmful sources. The rise of urbanization means more cars, buses, and trains, all of which contribute to an ever-increasing din. This urban soundscape is often relentless, with little respite for those living within it. In densely populated cities, people are exposed to high decibel levels, which can exceed safe thresholds for heart health. In many cases, the sheer volume of sound is not just unpleasant; it’s dangerous.
The solution is not as simple as reducing noise in our immediate surroundings, though efforts to reduce traffic noise and limit industrial sounds are essential. Some cities have taken steps to create quieter spaces by converting busy roads into pedestrian zones or installing noise barriers. These measures have shown to have a positive impact on public health, with research suggesting that even small reductions in noise can prevent premature deaths and improve overall well-being.
Credit: Opthalmology Journal
Doctors in India’s southern state of Tamil Nadu surgically removed a fully developed canine tooth from the upper eyelid region of a 16-year-old boy. They described the case as the first reported instance of an ectopic tooth in the upper part of the eye socket.
The case was reported by specialists at Aravind Eye Hospital in Madurai in the journal Ophthalmology.
The teenager visited the hospital with painless swelling of his left upper eyelid that had lasted a month. He had no history of injury to the area.
During the examination, doctors noticed two firm, whitish projections beneath the swollen area near the inner side of the upper eyelid, the experts shared in the paper.
A computed tomography (CT) scan revealed a dense structure resembling a tooth, with a crown and root, originating from the roof of the left eye socket. It extended towards the tissues in front of the orbital septum, a thin barrier separating the eyelid from the deeper eye socket, and protruded outwards with two tooth-like points on its surface.
The projections' appearance and texture raised the possibility that they were teeth. Dr. Meghana Tanwar, a medical consultant at Aravind Eye Hospital, told Live Science that they had a “fairly smooth surface and were hard on palpation.”
The doctors surgically removed the structure. Examination confirmed that it was a mature canine tooth without teratomatous elements, consistent with an ectopic tooth in the upper part of the eye socket.
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An ectopic tooth develops or emerges in an abnormal location. Some remain within the mouth but appear outside their usual positions in the dental arch, while others develop outside the normal arrangement of teeth.
The latter is particularly unusual. In this teenager's case, the tooth was located in the upper orbital region rather than within the normal dental arch.
Before confirming the diagnosis, doctors considered whether the projections could be associated with a teratoma, a type of tumor that can contain different tissues, including bone and tooth tissue. They also considered whether a foreign object might be lodged in the eyelid.
Imaging and examination of the removed structure helped establish that it was a mature tooth rather than tissue associated with a teratoma.
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Ectopic teeth have been documented in several locations, including the nasal cavity, chin and maxillary sinus, an air-filled cavity within the cheekbone. Previous reports have also described teeth in the lower part of the eye socket.
However, the doctors said they could not find an earlier report of an ectopic tooth in the superior orbit, the upper region of the eye socket.
“To the best of our knowledge this is the first and only ectopic tooth reported in the superior orbit,” Tanwar told Live Science.
“This was a first not just for us but the world over,” she said of the case. “We were very surprised by it.”
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The doctors could not determine why the tooth developed in this unusual location.
Possible explanations for ectopic tooth development include abnormalities during development, genetic factors, infections and physical trauma. However, the cause in this case remains unknown.
Imaging did not reveal any additional ectopic teeth. Tanwar said the likelihood of the teenager developing another such tooth was low.
Credit: athleticsweekly.com
Beth Dobbin, who represented Great Britain at the Tokyo Olympics in 2021, now spends more than 22 hours a day in bed.
The 32-year-old retired from athletics in April 2026, describing her illness as “serious” and “life-changing.” She was later diagnosed with myalgic encephalomyelitis (ME), a chronic condition with no known cure or specific treatment.
Dobbin reached the 200m semi-finals at the Tokyo Olympics and won bronze in the 4x200m relay with Scotland at the 2022 Commonwealth Games in Birmingham.
“It's life-decimating like I can't even put into words,” she said. “I feel like I've lost everything.”
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Dobbin’s health began deteriorating after back-to-back COVID-19 and glandular fever infections in the winter of 2021-22, the BBC
reported.
She experienced worsening fatigue, slower running times and soreness in the glands in her neck and armpits. Initial tests by British Athletics doctors failed to identify the cause.
Despite her symptoms, she continued training in 2022. At the European Championships, she false-started after losing all feeling in her legs while in the starting blocks.
Her condition subsequently worsened. She struggled to stand and spent most of her time in bed. After consulting four or five specialists, she was diagnosed with ME.
Dobbin has been confined to her house and bed since then. She gets out of bed only to eat and use the bathroom. Even conversations, bright lights or loud sounds can trigger a “crash” lasting days or, in severe cases, months. She has left the house just five times this year, the report said.
She now leaves home mainly for medical appointments and blood tests. Even conversations, bright lights and loud sounds can trigger a “crash” that lasts days or, in severe cases, months. She has left home just five times this year, according to the report.
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ME, also known as chronic fatigue syndrome (CFS), is a complex, chronic illness that can cause severe fatigue, pain, sleep disturbances and problems with thinking and memory. Symptoms do not improve simply with rest, and there is no established cure.
Dobbin rejects the term “chronic fatigue syndrome” because she believes it understates the severity of the condition.
“I wish it was just fatigue, that would be a walk in the park,” she said. “It's so much more than that. If I thought of fatigue in my top 20 symptoms it might just make the cut.”
She described the illness as feeling as though her entire body “is shutting down; it feels like you are a few days from death”.
More than 400,000 people in the UK are thought to have ME, although some estimates put the number above one million. The illness varies in severity, with some people continuing to work or study while others require extensive care.
Dobbin’s illness has also taken an emotional toll on her family.
“I'm just so desperate to help her but I can't because there is nothing. I'd take her anywhere, I'd take her to America if I had to, but there's nothing,” her father, Jim, said.
Dobbin describes her future as “very bleak” but hopes that greater awareness will lead to better treatment options.
The emergence of long COVID, which shares some symptoms with ME, has brought renewed attention to the illness and increased research funding.
She also wants to highlight the experiences of patients who are dismissed or disbelieved.
“A lot of people that have this illness that I've spoken to have said to me 'I don't get believed, people think I'm lazy'. I've never experienced one person that doesn't believe me and that's because, why would an athlete just suddenly stop after 20 years?”
Credit: iStock
Infections accounted for an estimated 2.3 million new cancer cases worldwide in 2024, or about 12% of all cancers diagnosed that year, according to a September 2026 study published in The Lancet Oncology. Among these, Helicobacter pylori (H. pylori), a bacterium that infects the stomach, was associated with an estimated 760,000 cancer cases.
A 2025 study by the International Agency for Research on Cancer (IARC), part of the World Health Organization (WHO), estimated that 76% of the 15.6 million stomach cancer cases expected over the lifetimes of people born between 2008 and 2017 could be attributable to H. pylori infection if prevention efforts do not improve.
Although infections can contribute to cancer development, they are not the only cause. Genetics, age and non-infectious factors also play a role, Dr. Vivek Kaul, Professor of Medicine in the Gastroenterology & Hepatology Division at the University of Rochester Medical Center, told HealthandMe in an exclusive interview.
“I think, generally speaking, I think H. pylori is a well-known carcinogen, mostly responsible for the development of gastric cancer,” Kaul said.
Is Cancer Increasingly Linked To Infections?
The findings highlight the contribution of infectious agents to the global cancer burden. However, Kaul cautioned against concluding that cancer is increasingly being driven by infections.
“But I don't think it's fair to say that, you know, we are moving towards a more infectious etiology for cancer,” he said.
“Cancer is a multifactorial disease state that develops, most commonly develops from genetic and from non-infectious causes,” Kaul added.
He explained that cancer development also depends on the ability of cells to defend themselves against external threats and genetic mutations.
“In many cases, cancer development comes primarily from genetic and age-related issues and boils down to the individual cell's ability, you know, to fight external attacks, such as from H. pylori type of agents or from genetic mutations,” he said.
Kaul also noted that other infectious agents have been implicated in cancer development.
H. pylori is a bacterium that infects the stomach lining and can persist for years. The infection can cause chronic inflammation and damage to stomach tissue, increasing the risk of gastric cancer.
According to scientists at the International Agency for Research on Cancer (IARC), part of the World Health Organization (WHO), H. pylori infection is responsible for an estimated 76% of stomach cancer cases worldwide. The infection often causes no noticeable symptoms, although some people may experience digestive complaints.
The bacterium can spread through contaminated food or water and close contact with infected individuals. In some cases, the infection can cause stomach ulcers. Because many infected people have no symptoms, testing may be needed to identify it.
The World Gastroenterology Organisation's global guidelines state that eradicating H. pylori before precancerous changes develop can help prevent gastric cancer. This evidence supports test-and-treat screening programmes for young adults in countries with a high disease burden and sufficient resources.
Testing can involve blood, breath or stool tests, depending on the clinical context. People diagnosed with the infection can receive treatment to eradicate the bacterium. Follow-up testing can then confirm whether treatment has worked.
Nobel laureate and gastroenterologist Dr Barry Marshall has also highlighted the potential for early treatment to reduce cancer risk.
“H. pylori is much more than an ulcer organism. If the infection persists for many years, it can cause chronic inflammation in the stomach and increase the risk of gastric cancer. That gives us an opportunity because this is a potentially preventable cause of cancer,” Marshall told PTI.
He stressed the need for wider testing, appropriate treatment and follow-up to ensure the infection has been cleared.
Beyond infection-associated cancers, Kaul highlighted the rising incidence of several digestive cancers.
“We know that esophageal squamous cancer is another example where we will be close to a million cases by 2035,” he said, pointing to the burden in East Asia, sub-Saharan Africa and South America, including India.
Kaul also identified liver cancer as a growing concern and noted the rise in colorectal cancer among younger people in Western countries. The increase in colorectal cancer among younger adults in the US contributed to recommendations to begin average-risk screening at age 45 rather than 50.
These trends underline the need to address infectious causes of cancer alongside other risk factors, while improving access to prevention, early detection and appropriate care.
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