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Anxiety is seen as an obstacle to success. It is a thought that always lingers in your mind before you start to do something. If you are doing something new or trying to do something that you have already done it in your life and have bad memories with. This thought creeps in. This thought that sometimes sends chills down your spine, makes you shiver, or that makes you feel like you cannot breathe, as if someone has put a huge rock on your heart is what defines anxiety. However, emerging research suggests that it can in fact, serve as a valuable tool for focus, motivation and problem solving. Studies on performance and stress, including the widely known Yerkes-Dodson curve, indicate that moderate levels of anxiety can enhance productivity, while both excessive and insufficient anxiety can hinder it.
The Yerkes-Dodson curve demonstrates that when anxiety is too high, it can become paralyzing, preventing individuals from taking action. On the other hand, very low anxiety can lead to complacency, reducing motivation and attention to detail. The ideal state falls somewhere in the middle, where anxiety signals importance, sharpens focus, and encourages preparation.
This perspective challenges the common belief that anxiety should always be suppressed. Instead, researchers argue that learning to harness anxiety can be beneficial. Studies have shown that people experiencing moderate anxiety often generate more creative solutions to problems compared to those who remain entirely calm.
Anxiety can serve as a sort of increased awareness, motivating people to take action in critical situations. Medical decision-making research show that concerned people are more likely to follow up on health issues, stick to treatment plans, and aggressively seek medical help. For example, studies on heart transplant patients show that people with moderate levels of anxiety are more likely to seek adequate care, which leads to improved survival rates.
The same strategy is used to address everyday difficulties. Anxiety tells the brain that something is important and requires effort. Anxiety can encourage you to be prepared and attentive, whether you're preparing for a presentation, making a critical life decision, or dealing with an uncertain situation.
Despite its potential benefits, anxiety can become debilitating when it causes what researchers term “functional impairment.” This occurs when excessive worry prevents individuals from fulfilling their daily responsibilities, whether in work, relationships, or personal well-being.
One method for distinguishing between helpful and harmful anxiety is cognitive reframing—shifting the perspective on stressors. Studies suggest that those who reframe anxiety as a performance-enhancing tool rather than a hindrance experience less distress and greater effectiveness in high-pressure situations. By viewing anxiety as a source of energy rather than a threat, individuals can improve their ability to navigate challenges.
Research on stress resilience indicates that experiencing a moderate amount of adversity in life can lead to stronger coping skills. Longitudinal studies on both humans and animals suggest that those who have faced and overcome moderate difficulties—such as personal loss or professional setbacks—tend to develop greater mental fortitude.
One study found that individuals who had endured a small number of adverse events exhibited better overall mental health than those who had either faced extreme hardship or had led relatively stress-free lives. This pattern aligns with the concept of "stress inoculation," where manageable levels of stress act as a form of psychological training, making individuals more resilient to future challenges.
Rather than attempting to eliminate anxiety, researchers suggest acknowledging it as a natural response that can be channeled productively. Psychological studies indicate that when people view their physiological responses—such as increased heart rate and adrenaline surges—as performance enhancers, their actual outcomes improve.
For instance, in high-stakes situations like public speaking or athletic competitions, individuals who interpret nervousness as a sign of readiness rather than fear tend to perform better. Physiologically, the body's response to excitement and fear is similar; the key difference lies in perception.
Research suggests that those who embrace a mindset of "anxiety as fuel" experience improved cognitive and physical performance. This approach aligns with studies showing that moderate stress prompts the release of hormones that enhance alertness and problem-solving ability, whereas overwhelming stress triggers a flood of cortisol, which can impair decision-making.
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Long COVID should be viewed as a web of overlapping symptoms rather than a single, uniform condition, according to a new systematic review published in eClinicalMedicine and reported by the Center for Infectious Disease Research and Policy (CIDRAP). The review highlights several recurring symptom patterns linked to long COVID, including neurological, respiratory, smell and taste-related, cardiopulmonary, and fatigue-driven clusters.
Researchers led by a team from Lanzhou University in Gansu, China, examined data from 64 studies conducted across 20 countries, covering nearly 2.4 million people. They grouped long COVID patients into subtypes using different approaches: symptom overlap in 30 studies, affected organ systems in 16 studies, symptom severity in nine, clinical markers in three, and other classification methods in the remaining research.
Among studies that focused on how symptoms appear together, fatigue stood out as the most consistently reported issue. It often occurred alone or alongside problems such as muscle and joint pain, brain fog, or breathlessness. Other symptom pairings that appeared frequently included loss of smell and taste, anxiety with depression, and various forms of musculoskeletal pain.
When researchers classified patients based on affected organ systems, respiratory problems were the most widespread, seen in about 47% of long COVID patients. Neurological symptoms followed at 31%, while gastrointestinal issues were reported by 28%. The authors stressed that these percentages reflect how often these clusters appeared within long COVID cases studied, not how common they are in the general population.
A smaller number of studies sorted patients by how severe their symptoms were, dividing them into mild, moderate, or severe categories using symptom scores, symptom counts, or quality-of-life measures. Three studies used clinical indicators for classification, including abnormal triglyceride levels and signs of restricted lung function on imaging.
The review also found that long COVID subtypes vary based on demographic, socioeconomic, and medical factors. Women were more likely to report fatigue and neuropsychiatric symptoms, while men more commonly experienced respiratory issues. Older adults tended to show higher rates of respiratory, cardio-renal, and ear, nose, and throat symptoms.
Racial and ethnic differences also emerged. Black and Hispanic individuals were more likely to experience respiratory, cardiac, and neuropsychiatric symptoms, whereas White patients showed higher rates of fatigue and musculoskeletal complaints.
COVID-19 variants appeared to influence symptom patterns as well. The researchers noted that the Alpha variant was closely linked to smell-related and respiratory symptoms, while the Delta variant raised the risk of ENT-related problems. In addition, higher body mass index, socioeconomic disadvantage, and existing conditions such as chronic obstructive pulmonary disease were strongly associated with cardiopulmonary symptom clusters and a heavier overall long COVID burden.
Overall, the findings reinforce that long COVID rarely affects just one system in the body. Instead, it tends to involve multiple overlapping symptom groups, pointing to the need for more tailored, patient-specific care.
The authors call for future studies to focus on creating standardized ways to classify long COVID, identifying the biological mechanisms behind different symptom clusters, and testing targeted treatments for specific subtypes. They note that this approach will be essential for moving toward precision medicine and improving outcomes for people living with long COVID.
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Singer and actor Ray J has reportedly been admitted to a hospital in Las Vegas after experiencing what were described as “heart pains,” according to TMZ. The 44-year-old was hospitalized on January 6, with sources telling the outlet that the episode was connected to a severe case of pneumonia. As of Thursday afternoon local time, Ray J was still receiving medical care, TMZ reported.
According to the report, doctors conducted multiple tests, including chest X-rays and an echocardiogram, an ultrasound that checks how well the heart is functioning. The findings from these tests have not been shared publicly. Neither Ray J nor his representatives have released an official statement addressing his health or confirming his current condition.
Although the initial concern focused on possible heart-related issues, TMZ noted that pneumonia appears to be the primary cause behind his hospitalization. It is still unclear whether Ray J remains admitted or has since been discharged.
This is not the first time Ray J has dealt with a serious pneumonia-related health scare. In late 2021, he was hospitalized after doctors initially suspected COVID-19 before later confirming pneumonia.
At the time, Ray J described the ordeal as life-threatening. “I thought it was over,” he told TMZ while recovering. “I started praying and knew God is good, so I just kept praying.” He later made a full recovery and was discharged after receiving oxygen support.
Despite reports of his recent hospitalization, Ray J has remained active online. He recently posted on Instagram to promote an upcoming event in Austin, Texas, scheduled for January 18 as part of his birthday celebrations. The post made no reference to any health concerns.
Pneumonia is an infection of the lungs that causes inflammation in the air sacs, known as alveoli. These air sacs may fill with fluid or pus, making breathing difficult and reducing the amount of oxygen reaching the bloodstream. The illness can be caused by bacteria, viruses, or fungi and often presents with symptoms such as coughing, fever, chills, and shortness of breath.
While pneumonia is usually treatable with medication, it can become severe in certain cases, particularly among older adults, young children, and people with weakened immune systems. Because of these risks, pneumonia remains a leading cause of hospitalization, according to the Cleveland Clinic.
Yes. Pneumonia can trigger heart-related symptoms and complications. According to Healthline, patients may experience a rapid heart rate, chest pain, or increased strain on the heart. In more serious cases, pneumonia can worsen existing heart conditions or lead to complications such as arrhythmias, heart failure, or even heart attacks.
The intense inflammation caused by a lung infection places added stress on the cardiovascular system, making early diagnosis and treatment especially important, particularly for older adults or those with underlying heart disease.
Pneumonia is a common illness and is frequently linked to heart complications. Research cited by the National Institutes of Health shows that roughly one in four hospitalized adults with pneumonia develop major cardiac issues, including heart attacks, heart failure, or abnormal heart rhythms. These complications significantly increase the risk of death.
Heart disease also raises the likelihood of developing pneumonia, creating a two-way relationship between the two conditions. Inflammation from the infection can put additional pressure on the heart, especially in older patients and those with pre-existing cardiovascular problems.
Yes, pneumonia accompanied by heart-related symptoms can be fatal. Cardiac complications are common and greatly increase mortality risk, particularly among older adults and individuals with existing heart conditions such as heart failure. The danger is highest in the period immediately following the infection.
Pneumonia places heavy stress on the heart and can trigger heart attacks, strokes, or a rapid worsening of existing heart disease, underscoring the importance of prompt medical care.
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Performing short and intense exercises that last for about 10 minutes may significantly reduce the risk of colorectal cancer, a British study shows.
Researchers at Newcastle University have found that completing brief workouts increases the concentration of several small molecules in the blood that have previously been linked to reducing inflammation, improving blood vessel function and metabolism.
The findings, which were published in the International Journal of Cancer, also noted that even short bouts of exercise can influence the activity of genes that govern tumor growth and fight against cancer.
Dr Sam Orange, Senior Lecturer in Clinical Exercise Physiology at Newcastle University and lead study author, said of the results, "What’s remarkable is that exercise doesn’t just benefit healthy tissues, it sends powerful signals through the bloodstream that can directly influence thousands of genes in cancer cells.
“It’s an exciting insight because it opens the door to find ways that mimic or augment the biological effects of exercise, potentially improving cancer treatment and, crucially, patient outcomes.
“In the future, these insights could lead to new therapies that imitate the beneficial effects of exercise on how cells repair damaged DNA and use fuel for energy.”
Researchers had asked each volunteer to complete a short, intense cycling test that lasted about 10 minutes and collected each of their blood samples which were then meticulously analyzed.
After studying about 249 proteins, the experts found nearly 13 protein had increased in their blood flow after the short exercise including interleukin-6 (IL-6), which helps repair the DNA of damaged cells.
When these exercise-induced proteins were applied to colorectal or bowel cancer cells by the scientists in a lab, they discovered that these new proteins could alter the activity of over 1,300 genes, particularly those who were responsible for DNA repair, energy production and cancer cell growth.
As a result, they concluded that performing even small exercises can activate a set of genes in the body's cells that can support efficient use of oxygen and promote energy metabolism. Additionally, the British researchers also discovered that the cell genes which have been previously associated with rapid cell growth were switched off after working out, suggesting that this might aid in controlling cancerous growths in the body.
Colorectal or bowel cancer is a common cancer that forms in the tissues of the colon (large intestine) or rectum, often starting as non-cancerous growths called polyps that turn cancerous over time.
Despite being treatable and preventable, colorectal cancer is currently the second leading cause of cancer-related deaths worldwide. Over 70,000 new cases of colorectal cancer occur annually in India, making it the fourth most common cancer in the country.
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