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Have you ever been in such a deep slip and ended up waking coughing? Chances are, you choked on your own saliva. Yes, it is very much possible. Saliva is a clear liquid produced by your salivary glands that play a crucial role in digestion and oral health. This is because it washes away bacteria and food particles. The body on an average produces about 1 to 2 litres of saliva daily, and a lot of it is swallowed unconsciously. However, when saliva does not flow smoothly down the throat, it can lead to choking, which can be uncomfortable.
Here's all that you need to know about what causes it, and how can you prevent it.
This happens when the muscles that are responsible for swallowing weaken or malfunction due to health issues and the symptoms may include:
Some of the common reasons include:
Acid reflux occurs when stomach acid travels back into the esophagus and mouth, irritating the esophagus and increasing saliva production to neutralize the acid. This buildup of saliva can lead to choking.
Acid reflux can be diagnosed via endoscopy or X-rays. Treatment includes antacids to reduce stomach acid.
When you are sleeping, some abnormal swallowing can occur during that time and it may cause saliva to pool in the mouth and flow into the lungs. This is what leads to choking. This condition is often linked to obstructive sleep apnea (OSA), which causes breathing to pause during sleep.
CPAP machines to provide continuous airflow
Oral mouth guards to keep the airway open
Growths in the throat, whether benign or cancerous, can narrow the esophagus and impede swallowing.
Treatment may involve surgery, radiation, or chemotherapy, depending on the nature of the growth.
Dentures can confuse the brain into perceiving them as food and it may trigger excess saliva production and cause choking. The best way to work this out is by consulting a dentist for proper fitting and adjustment of dentures.
Conditions like Parkinson’s disease and ALS can impair the nerves that control swallowing.
Doctors may prescribe medication to reduce saliva production or teach swallowing techniques.
Excessive alcohol consumption can relax throat muscles, allowing saliva to pool and cause choking.
Moderate alcohol intake and sleep with your head elevated.
Speaking continuously without pausing to swallow can lead to saliva entering the windpipe.
Take pauses to swallow while talking.
Thickened saliva or mucus due to allergies or infections can obstruct the throat, especially during sleep.
Antihistamines, cold medication, or antibiotics may be prescribed, depending on the underlying cause.
In case of sleep apnea, sleep with your head elevated or on your side. You can also try and avoid lying flat right after eating and eat smaller meals to prevent acid reflux. It is also important that you sip water throughout the day to clear any saliva buildup. In severe cases, use over-the-counter medicines for allergies or consult a healthcare provider.
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It has been long suspected that Western diets could contribute to the risk of colorectal cancer. The theory may finally have a clearer scientific explanation.
A new study suggests that gut bacteria can convert compounds produced by high-fat, low-fibre diets into cancer-causing chemicals. The research sheds light on how unhealthy eating habits may lead to cancerous tumour growth in colon in the long term.
Published in the journal Gut, the research was led by Dr. Annika Osswald (first author) and Dr. Soeren Ocvirk (corresponding author), along with a large team of international scientists.
The collaboration included researchers from Technical University of Munich (TUM), Germany, German Institute of Human Nutrition (DIfE), RWTH Aachen University, Freie Universität Berlin, University Hospital of Regensburg, and other institutions.
Researchers found that a western-style diet, which is commonly high in red and processed meat, saturated fats, refined carbohydrates and ultra-processed foods, significantly changes the composition of the gut microbiome.
These altered bacteria then modify bile acids in ways that promote inflammation and create an environment that accommodates the development of colorectal cancer tumours.
According to the researchers, diet alone is not the only factor. The trillions of microbes living in the intestine determine how food is processed, producing metabolites that can either protect the gut or damage it.
The study found that specific bacterial groups transformed bile acids into compounds that stimulated tumour growth in the colon.
This provides one of the strongestt explanations yet for why western dietary patterns have consistently been associated with higher risk of colorectal cancer.
"Our findings highlight the critical interaction between diet, gut microbes and cancer biology," the researchers noted, adding that targeting the microbiome could become a future strategy for preventing colorectal cancer.
Also read: Why English Actor Peter Duncan Is Advocating For Focal Therapy After His Prostate Cancer Treatment?
A western diet typically includes:
Previous research has repeatedly linked this eating pattern with obesity, diabetes, heart disease and colorectal cancer, but scientists have long found it challenging to explain the exact cause until now.
The human gut is home to trillions of bacteria that help digest food, regulate immunity and produce beneficial compounds like fatty acids.
A fibre-rich diet supports good bacteria that reduce inflammation, whereas diets high in fat and processed foods can cause microbial imbalance.
The new findings suggest this imbalance changes how bile acids are metabolised, increasing the production of molecules capable of damaging the colon and supporting cancer growth in the long run.
Also read: WHO Cancer Agency Flags 3 Common Medicines As Carcinogenic: What It Means For Millions Of Patients
Earlier research has linked harmful gut bacteria, including toxin-producing E. coli, with DNA damage that may begin early in life and contribute to the rise of colorectal cancer among younger people.
However, the latest findings do not prove diet alone causes cancer. Genetics, obesity, physical inactivity, sedentary life, smoking and alcohol consumption also influence risk.
However, they say maintaining a fibre-rich diet with fruits, vegetables, legumes and whole grains may help preserve a healthier gut microbiome and lower long-term colorectal cancer risk.
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Could air pollution may do more than harm the lungs and heart? A new study has found that exposure to polluted air could trigger painful flare-ups in people living with rheumatoid arthritis (RA).
The study comes when evidence is mounting that environmental factors contribute significantly to autoimmune diseases.
The study, published in the Annals of the Rheumatic Diseases, found that excessive exposure to air pollutants, particularly fine particulate matter (PM2.5), was associated with increased rheumatoid arthritis activity and a greater risk of flare ups.
Researchers say the findings suggest that improving air quality should become an important part of managing the chronic condition, alongside treatment, medications and lifestyle changes.
"Our findings highlight that environmental exposure, especially air pollution, may significantly influence rheumatoid arthritis disease activity and flare risk," the researchers said, noting that patients and clinicians should consider air quality as a modifiable risk factor.
Also read:
Rheumatoid arthritis is an autoimmune disease in which the immune system attacks healthy joints, causing pain, swelling, stiffness and, over time, permanent joint damage.
While genetics, smoking and infections have been recognised as risk factors, scientists are investigating how environmental pollutants may worsen the disease.
The latest findings are particularly relevant for countries such as India, where millions are exposed to unhealthy air for large parts of the year. Previous reports have already linked poor air quality in cities like Delhi to rising concerns over autoimmune diseases.
Also read: Severe COVID-19 Can Reactivate Dormant Viruses, May Fuel Long COVID Symptoms: Study
A flare is a period when rheumatoid arthritis symptoms suddenly worsen. During this time, people may experience:
Flares can last from a few days to several weeks and are often triggered by infections, stress, missed medications or other environmental factors.
Researchers believe tiny airborne particles like PM2.5 can enter the lungs and bloodstream, triggering inflammation throughout the body.
This inflammatory response may overstimulate the immune system, making rheumatoid arthritis symptoms worse and increasing the likelihood of painful flare-ups.
Also read:
The researchers emphasised that the study shows an association rather than proving that air pollution directly causes rheumatoid arthritis flares.
However, the consistent link suggests reducing exposure to polluted air may help lower the chances of flare-ups in high-risk individuals.
Experts advise patients to continue prescribed medications, constantly monitor local air quality, avoid outdoor activities during periods of severe pollution when possible, and discuss symptom changes with their rheumatologist.
The findings add to a growing body of research linking air pollution with autoimmune diseases.
Earlier studies have suggested that long-term exposure to pollutants may increase the risk of developing rheumatoid arthritis, while the new research indicates polluted air may also worsen symptoms in people already living with the disease.
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Commonly prescribed medicines for high blood pressure and heart disease could also help antidepressants work better, according to a new study by researchers at the All India Institute of Medical Sciences (AIIMS).
The research offers hope for people with depression whose condition is not adequately managed with standard antidepressants.
The study found that certain cardiovascular medications, when taken along with antidepressants, may improve the brain's response to treatment, potentially making clinical depression treatments more effective.
The findings establish a foundation for repurposing common medicines to improve mental health treatment. The researchers stress that larger clinical trials are needed to validate the study before it can be included in standard depression treatments.
"Our findings suggest that some drugs already being used to treat hypertension and heart disease may have the potential to act as adjuncts to antidepressant therapy," the AIIMS research team said.
They also said that the strategy could particularly benefit patients who do not respond to antidepressants well.
Even though depression affects hundreds of millions of people worldwide, almost one-third of patients do not receive adequate relief from their first antidepressant.
Even when medications work, they often take several weeks to show results. This has led researchers to explore more ways to improve treatment outcomes, especially for mental health issues.
Repurposing existing medicines is considered an attractive strategy because their safety profiles are already well understood, potentially reducing the time and cost needed to bring new treatment options into clinical practice.
Also read: Mental Health Crisis In Israel Military: Why Suicides Among Soldiers Are Increasing?
Antidepressants, including selective serotonin reuptake inhibitors (SSRIs), are among the most commonly prescribed medicines for moderate to severe depression. They work by changing the levels of neurotransmitters like serotonin in the brain, helping regulate mood over time.
However, individual responses may vary widely, and many patients end up needing multiple treatment approaches before achieving any relief.
While the exact biological mechanisms are still under investigation, researchers believe that some cardiovascular drugs may have an effect on pathways associated with inflammation, blood flow to the brain, or nerve signaling, all of which are increasingly play a role in depression treatment.
The researchers noted that these medicines are not intended to replace antidepressants. Instead, they could eventually be used alongside standard treatment to improve its effectiveness in some patients.
Experts caution that patients should not start or stop blood pressure or heart medications in the hope of treating depression without clinical supervision.
More clinical studies are required to identify which cardiovascular drugs offer the greatest benefit, which patients are most likely to respond, and whether the combination remains safe over long-term use.
The AIIMS study could pave the way for a cost-effective strategy to improve depression treatment by giving a new role to medicines that are already widely available.
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