Alarming Reality Of Extreme Drinking On Holidays And Occasions
With the holiday season high, there is festive cheer, family gatherings and also an undeniable increases in alcohol consumption that fills the air. Christmas and New Year's Eve celebrations to spring break and bachelor parties and sporting events that bring together huge crowds for celebrations mean that drinking becomes synonymous with partying. But behind the revelry lies a much darker behavior: high-intensity drinking.
Alcohol is the most widely used substance in the United States; it has been reported that 84% of adults aged 18 and older reported lifetime use. Moderate drinking is socially acceptable, but high-intensity drinking is an alarming trend. The behavior of consuming eight or more drinks over a few hours for women and 10 or more for men exceeds binge drinking and significantly increases risk for harm.
High-intensity drinking is far from being just a mere passing concern; it is instead a public health crisis. The burden is even greater as 29 million people in the United States suffer from alcohol use disorder. That has caused over 140,000 deaths annually while accounting for 200,000 hospitalizations and 7.4% of visits to emergency departments in the United States. However, only 7.6% of these affected get treated, thus forming a glaring gap in handling this concern.
High-intensity drinking is a dangerous escalation from traditional binge drinking, characterized by consuming double or triple the standard binge amounts. While binge drinking involves four or more drinks for women and five or more for men, high-intensity drinkers often surpass these levels, leading to blood alcohol concentrations (BAC) exceeding 0.2%—a level that significantly impairs judgment and motor skills.
According to Dr. George Koob, the director of the National Institute on Alcohol Abuse and Alcoholism (NIAAA), high-intensity drinking is one of the factors that intensify the risks of injuries, overdose, and death. It is also very highly associated with the onset of AUD, since the chance of addiction increases with increased alcohol consumption per occasion.
One of the most troubling consequences of high-intensity drinking is alcohol-induced blackouts, periods of amnesia where individuals may appear functional but are incapable of forming memories. Blackouts occur when alcohol disrupts the hippocampus, the brain region responsible for memory formation.
Blackouts are often categorized into two types:
1. Fragmentary Blackouts: Characterized by spotty memory, where recalling certain details can trigger partial recollection.
2. En Bloc Blackouts: Significant amnesia for hours, wherein no memory is created at all, even if tried to be recalled.
Aside from memory loss, intense binge drinking is linked with poor decision-making, violence, injury, and conflicts in personal relationships.
Holidays and celebrations create the perfect storm for high-intensity drinking. According to research, adults drink nearly double the amount of alcohol during holidays like Christmas and New Year's Eve than they do at any other time of the year. It is during these periods of social gathering, holiday stress, and seasonal sadness that people drink in excess.
For college students, experiences like spring break and 21st birthdays increase the danger. Some studies indicate that students, especially those who travel with buddies to spring break, indulge in more alcohol and make more serious decisions than any student who remains at home or goes with their family to other destinations. Sporting events are, too, notorious for promoting drunk consumption, especially among male customers. Alcohol consumption usually goes high during Super Bowl Sunday, thus leading to games day violence and arrests.
High-intensity drinking impacts not only physical health and mental well-being but also social relationships.
- Alcohol poisoning
- Severe dehydration and electrolyte imbalances
- Hypoglycemia
- Risky sexual behavior
- Injuries and accidents
- Liver damage, alcoholic hepatitis, and cirrhosis
- Cardiovascular diseases such as arrhythmias and cardiomyopathy
- Neurological damage, including memory deficits and blackouts
- Progression to alcohol dependence or AUD
High-intensity drinking is strongly linked with increased risks of depression, anxiety, and suicidal ideation. Poor decision-making during episodes can lead to long-lasting consequences, including damaged academic, professional, or personal outcomes.
Combating high-intensity drinking requires education, early intervention, and accessible treatment options. The NIAAA has defined high-intensity drinking to be distinct from binge drinking and has called for targeted approaches to decline prevalence and associated harms.
One promising treatment option is naltrexone, which a medication helps control alcohol cravings. Encouraging in preliminary evidence, more extensive clinical trials will be necessary to ascertain its efficacy more specifically in high-intensity drinkers.
As we head into the holiday season and other special occasions, it is important to heighten awareness of the dangers of high-intensity drinking. A good understanding of long-term consequences and seeking help when alcohol-related issues arise can be the difference between life and death. Celebrations should be about joy and connection, not about the gateway to harm.
If you or someone you know drinks at dangerous levels or have an alcohol use disorder, there is help available. Remember, for suspected cases of alcohol poisoning, dial 911. In this way, we can foster healthier relationships with alcohol and create safer environments for everyone.
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Higher education has changed far more than we often acknowledge. Universities are no longer simply places where students earn degrees. They are where young adults spend some of the most formative years of their lives, away from familiar support systems, making independent decisions, navigating uncertainty and, for many, encountering the first signs of a mental health condition. That quiet shift has expanded the role of educational institutions in ways that were never envisaged a decade ago.
The conversation around student wellbeing has evolved alongside this change. Mental health is no longer viewed as a subject to be discussed only after a crisis. Students are speaking more openly, parents are asking different questions, faculty members are becoming more aware and institutions are recognising that emotional wellbeing is closely linked to learning, participation and long term outcomes. This change deserves to be welcomed because it has helped move mental health from the margins of campus life to the centre of institutional responsibility.
Yet one assumption continues to shape much of this conversation. The presence of a counsellor is often seen as evidence that a campus is equipped to support student mental health. Counsellors remain indispensable and, for many students, they provide exactly the support that is needed. The challenge arises when counselling is expected to fulfil every role within a mental healthcare system.
Every effective healthcare system is built on layers of expertise. A physician does not replace a surgeon. A laboratory does not replace a diagnosis. Emergency care does not replace rehabilitation. Mental healthcare should be viewed no differently. Counselling is often the first point of contact, but the needs of students do not end there.
Some require structured therapy, others psychiatric evaluation, some ongoing clinical monitoring, and a few immediate crisis intervention. A mature campus mental healthcare system should be equipped to respond across this entire spectrum. The conversation should therefore move beyond whether campuses have counsellors to whether they have a system capable of supporting every stage of care.
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Encouragingly, public policy is beginning to recognise this changing reality. The University Grants Commission’s draft guidelines on mental health and wellbeing for higher educational institutions, with recommendations on counsellor ratios, dedicated wellbeing centres, round the clock helplines and mechanisms for early identification of distress, mark an important step in strengthening institutional support. More importantly, they open the door to a broader conversation on what comprehensive mental healthcare within higher education should look like over the coming years.
Building such a system requires looking beyond individual appointments. One of the most valuable lessons from developing healthcare services is that outcomes are rarely determined by the first consultation alone. They depend on how seamlessly care continues afterwards. If a counsellor recognises that a student needs specialised assessment, how quickly can that happen?
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If medication becomes necessary, is psychiatric care available without delay? If a student experiences a crisis outside campus hours, is there a clearly defined pathway to immediate support? If treatment begins, who ensures continuity during semester breaks or after the student returns home? These are not administrative questions. They are questions that shape recovery.
A comprehensive campus mental healthcare ecosystem should therefore bring together different levels of expertise rather than rely on one profession alone. Counsellors, clinical psychologists, psychiatrists, experienced mental health specialists and emergency support services each play a distinct role. Their contribution becomes most effective when they work as part of an integrated network with clear referral pathways, shared clinical responsibility and continuity of care that extends beyond the physical boundaries of the campus.
Also read: Sugar Rationing In First 1,000 Days Linked To Lower Depression, Cancer Risk In Adulthood
Equally important is recognising that mental healthcare should not begin only after a student seeks help. Institutions have long understood the value of preventive healthcare through regular physical health assessments, vaccination drives and awareness programmes. Mental health deserves the same thoughtful approach.
Periodic, voluntary mental health check ins, appropriate screening, trained faculty and peer support networks, backed by specialist expertise, can help identify concerns early while respecting privacy, dignity and informed consent. Early recognition is not about labelling students. It is about ensuring that support reaches them before distress becomes disabling.
Technology can strengthen this ecosystem, but it cannot replace it. Digital consultations, secure follow up, coordinated records and access to specialists across locations can make care more continuous, particularly for students studying away from home. Their real value lies in connecting different parts of the system rather than functioning as isolated solutions.
The quality of a university has traditionally been measured through its academic standards, faculty and research. Increasingly, it will also be measured by how well it supports the people who make learning possible. Mental healthcare deserves to be seen as part of that institutional foundation, not as an additional welfare service that sits alongside education.
The conversation has already moved beyond whether student mental health matters. The next step is to recognise that no single professional, however skilled, can meet every need that students may bring with them. Every campus needs more than a counsellor because every student deserves access to a mental healthcare system that is prepared not only to listen, but also to respond, support and care through every stage of that journey.
By Dr. Jothi Neeraja, Founder, Chairwoman and Managing Director, Maarga Mindcare
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Anxiety and depression are major concerns in adulthood, but could nutrition during the earliest stages of life influence health decades later? Two new studies suggest that lower sugar exposure during the first 1,000 days of life may be associated with lower risks of anxiety, depression and even several cancers later in life.
The findings, from studies published in Translational Psychiatry and PNAS point to the potential long-term effects of early-life nutrition. However, the findings do not mean that restricting sugar in infancy directly prevents these diseases.
A 2026 study led by researchers from the University of Surrey, UK, analyzed 46,448 people born between October 1951 and March 1956.
Participants were grouped according to how long they were exposed to sugar rationing: from in utero only to 24 months. People conceived after food rationing had ended served as the main comparison group.
Researchers also analyzed brain MRI data from 5,990 participants.
Compared with people conceived after rationing ended, those exposed to sugar rationing for the three longest periods had significantly lower hazards of both anxiety and depression.
When later-life sugar intake was considered, the association with anxiety persisted among those exposed throughout pregnancy and the first two years of life, while the association with depression weakened and was no longer statistically significant.
Further, MRI analysis found differences across rationing groups in 80 of 139 gray matter regions. Analysis identified 11 regions that differed from participants conceived immediately after rationing ended, including the brainstem, occipital fusiform gyrus and several cerebellar regions.
A separate study published in PNAS examined whether sugar exposure during the first 1,000 days could influence cancer risk later in life.
Researchers from China Agricultural University and the University of Cambridge used the abrupt end of UK sugar rationing in September 1953 as a natural experiment. The analysis included 64,761 UK Biobank participants born between 1951 and 1956.
Compared with those whose first 1,000 days were not affected by rationing, participants exposed to rationing showed lower incidence of several cancers:
The researchers identified two possible pathways behind the association.
Sugar is an important source of energy for the brain, and very low blood glucose can impair brain function. However, excess sugar intake has also been associated with metabolic problems that can affect long-term health.
When it comes to cancer, sugar does not directly cause cancer, and cutting out all sugar does not “starve” cancer cells. Both healthy and cancer cells use glucose for energy.
However, consistently consuming excessive amounts of added sugar can contribute to weight gain and obesity, which are established risk factors for several cancers.
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Men who consume higher amounts of dietary nitrite may have a greater risk of colorectal cancer, particularly the type affecting the distal colon, according to a new study published in the Journal of the National Cancer Institute.
The study followed 82,009 middle-aged and older adults in Sweden for more than two decades and identified 3,170 cases of colorectal cancer.
Researchers found that men in the highest category of nitrite intake had a 23% higher risk of colorectal cancer overall compared with those in the lowest category. The strongest association was for for distal colon cancer, where the risk was 50% higher.
The findings were not seen in women, and dietary nitrate was not associated with colorectal cancer in either men or women.
Researchers from Karolinska Institutet in Sweden used dietary information collected in 1997 and updated in 2009 and 2019. They linked this information with Sweden's cancer registry to identify colorectal cancer cases through 2022.
Rather than relying only on participants' diet at the beginning of the study, researchers used repeated measurements to capture changes in nitrite and nitrate intake over time. Among men, the highest versus lowest levels of nitrite consumption were associated with a hazard ratio of 1.23 for colorectal cancer. For distal colon cancer, the hazard ratio rose to 1.50.
Also read: How Genetic and Genomic Testing Can Help Women Make Smarter Cancer Decisions
Nitrite and nitrate are related compounds, but they are found in different foods and behave differently in the body. Nitrate occurs naturally in vegetables, particularly leafy greens, as well as drinking water. Nitrite is also naturally present in some foods but is commonly used as a preservative in processed meat and other animal products.
The researchers found no association between dietary nitrate and colorectal cancer, making the distinction particularly important.
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A possible explanation involves N-nitroso compounds, which can form when nitrate and nitrite undergo chemical reactions in the body. Some N-nitroso compounds are known to be carcinogenic in animals.
The researchers saw that previous studies have also investigated possible links between nitrite exposure and cancers of the gastrointestinal tract. However, the exact biological mechanism behind the association observed in this study remains uncertain.
The researchers also found that the association was specific to men, and they said the reason for this difference is unclear.
The authors noted that the higher risk associated with nitrite intake was confined to men and suggested several possible biological explanations, including differences in oxidative stress and other sex-specific factors.
But they stressed that these mechanisms have not been established as the reason for the finding. The study concludes that further research should examine nitrite and colorectal cancer separately in men and women and.
The study was observational, meaning researchers identified an association between dietary nitrite intake and cancer risk but could not prove a direct cause and effect relationship.
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