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Low-dose CT chest scans could help detect pneumonia in at-risk patients while exposing them to only small amounts of radiation, a new study has found. The research, published in Radiology: Cardiothoracic Imaging, shows that ultra-low-dose scans can effectively detect pneumonia in patients with compromised immune systems, enabling doctors to treat the infection before it becomes life-threatening. According to the researchers, these scans expose patients to just 2% of the radiation dose used in a standard CT scan.
"This study paves the way for safer, AI-driven imaging that reduces radiation exposure while preserving diagnostic accuracy,” lead researcher Dr Maximiliano Klug, a radiologist with the Sheba Medical Center in Ramat Gan, Israel, said in a news release. He added that CT scans are the gold standard for detecting pneumonia but there are concerns regarding the risk posed by repeated exposure to radiation. There is a solution- ultra-low-dose CT scan. However, the problem is that these scans can be grainy and hard to read, researchers said.
Study Gives Solution To This
To overcome that, Klug's team developed an AI program that could help "de-noise" low-dose scans, making them sharper and easier to read. Between September 2020 and December 2022, 54 patients with compromised immune systems who had fevers underwent a pair of chest CT scans -- a normal dose scan and an ultra-low-dose scan. The AI program cleaned up the low-dose scan, and then both sets of images were given to a pair of radiologists for assessment. Radiologists had 100% accuracy in detecting pneumonia and other lung problems with the AI-cleaned low-dose scans, but 91% to 98% accuracy in examining the scans that hadn’t been improved through AI, results show.
"This pilot study identified infection with a fraction of the radiation dose," Klug said. "This approach could drive larger studies and ultimately reshape clinical guidelines, making denoised ultra-low dose CT the new standard for young immunocompromised patients.
How Can You Detect Pneumonia?
Pneumonia is a lung infection that causes the air sacs in the lungs to fill with fluid or pus and can be caused by bacteria, viruses, or fungi. The symptoms can range from milk to severe, which includes:
Coughing with or without cough
Fever
Chills
Trouble breathing
Chest pain, especially when breathing deeply or coughing
Sweating or chills
Rapid heart rate
Loss of appetite
Bluish skin, lips, and nails
Confusion.
How to detect Pneumonia in coughing newborns and toddlers?
Pneumonia can severely affect newborns and young children as their lungs are comparatively more sensitive. As per Dr Goyal, young children can cough for various reasons including seasonal infections and tonsillitis, which is very common in this age group. But if they look visibly irritable and have poor sleep patterns, then parents must reach out to an expert. "I am not saying that parents must visit a hospital but any local paediatrician would be able to detect pneumonia in your kid.
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Veteran Republican Senator Lindsey Graham, one of President Donald Trump's closest allies, has died at the age of 71. As per the preliminary findings released by the District of Columbia's medical examiner, he passed away after suffering an aortic dissection.
His office said, “On the evening of Saturday, July 11, Senator Lindsey Graham passed away after a brief and sudden illness. Senator Graham's family appreciates prayers at this time and asks for privacy during this incredibly difficult period.”
Graham was a prominent South Carolina Republican and former Air Force lawyer who served in Congress for more than three decades. He had turned 71 years old just two days before his death.
In a statement on Truth Social, US President Donald Trump wrote: "Melania and I are deeply saddened by the sudden passing of Senator Lindsey Graham. Lindsey was a true American Patriot, one of the best people and Senators I have ever known. He was always there when our Nation needed him, and he gave everything he had to the people of South Carolina and the United States. We will miss him greatly. Our prayers are with his family, friends, and everyone who loved this truly great man."
The preliminary medical examiner’s report later confirmed that the underlying cause was aortic dissection due to arteriosclerotic cardiovascular disease, commonly known as the hardening of the arteries.
According to reports, a final death certificate will be issued after additional toxicological and microscopic testing.
Graham’s passing has sparked discussion about aortic dissection, a rare but extremely dangerous cardiovascular emergency that requires immediate medical treatment.
An aortic dissection occurs when a tear develops in the inner layer of the aorta, the body's largest artery that carries blood from the heart to the rest of the body. Blood surges through this tear, forcing the layers of the artery wall apart, effectively “dissecting” them.
When the wall of this aorta tears, blood flow to vital organs can become compromised, and the weakened artery may rupture, often leading to a fatal internal bleeding.
Cardiologists consider aortic dissection to be one of the most life-threatening cardiovascular emergencies. The condition can rapidly lead to complications like:
The risk of death significantly increases if the condition is not diagnosed and treated quickly.
There are several causes of aortic dissection. Major risk factors include hypertension, atherosclerosis, smoking, high cholesterol, connective tissue disorders like Marfan syndrome or Ehlers-Danlos syndrome, congenital abnormalities of the aortic valve or previous heart surgery
In Graham's case, preliminary findings stated that the aortic dissection was caused due to arteriosclerotic cardiovascular disease, indicating that long-term hardening of the arteries likely contributed to the fatal tear.
An aortic dissection often begins suddenly. As the symptoms of an aortic dissection can mimic those of a heart attack, immediate medical evaluation is essential. Symptoms may include:
According to experts, the risk of aortic dissection can be reduced by making several lifestyle changes early on:
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For many people with COPD, vigorous exercise can feel impossible because of breathlessness and fatigue. However, a latest study suggests that replacing sedentary time with light physical activity could still have substantial health benefits.
People living with chronic obstructive pulmonary disease (COPD) may not need intense workouts to improve their longevity anymore.
A new study involving more than 800 adults with COPD has found that even light physical activity, like walking, doing household chores, or simply spending less time sitting, was linked to a significantly lower risk of early death.
The findings offer hope for millions of people with COPD, a progressive lung disease that makes breathing difficult, discouraging patients from being physically active. This study highlights an important message for COPD patients: every bit of movement matters.
After analyzing data from more than 800 participants, researchers found that those who spent more time engaged in light-intensity activities had better long-term survival than those who were largely inactive. The association remained even after accounting for factors including age, smoking history, disease severity, and other medical conditions.
"Our findings suggest that even modest increases in light physical activity may improve long-term survival in patients with COPD," the researchers said.
They added that encouraging patients to reduce the amount of time they spend sitting may be a realistic goal, especially for those who are unable to perform moderate or vigorous exercise.
Examples of light physical activity include slow walks around the neighborhood, gardening, light housework, standing while talking on the phone, or taking frequent movement breaks throughout the day. While these activities may seem small, researchers say they can add up to meaningful health benefits over time.
COPD, which includes emphysema and chronic bronchitis, is one of the leading causes of illness and death worldwide. The disease gradually damages the lungs, making everyday activities increasingly difficult.
As symptoms worsen, many patients become less active, creating a cycle that can lead to weaker muscles, poorer physical function, and an even greater loss of independence.
The new findings promotes existing recommendations from respiratory health experts, who have always pushed people with COPD to remain as active as their condition safely allows.
Regular movement has been shown to improve muscle strength, reduce breathlessness during daily activities, enhance energy levels, support better sleep, and improve overall quality of life.
"This study reinforces the idea that every movement counts," experts say. Instead of focusing only on structured exercise sessions, patients may benefit from finding simple ways to move more throughout the day.
However, researchers caution that the study found an association rather than proving that light physical activity directly causes longer survival.
For people living with COPD, the takeaway is that improving health does not always require intense workouts. Sometimes, taking a few extra steps, standing up more often, or staying gently active throughout the day may be enough to make a meaningful difference and improve the quality of life.

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A new study has found out what researchers call the strongest evidence yet that long COVID may directly injure the brain's dopamine system, offering an explanation for symptoms like fatigue, brain fog, poor memory, slowed movement, and lack of motivation that persist long after the initial infection.
While it has been a long time since the end of the COVID pandemic, its effects continue to linger even today. Several patients who contracted COVID continue to suffer. This is known as long COVID.
Dopamine is a neurotransmitter that plays a key role in movement, motivation, learning, reward, and memory.
The study, conducted by researchers at the Centre for Addiction and Mental Health (CAMH) in Toronto and published in the journal eBioMedicine, used advanced positron emission tomography (PET) scans to examine the brains of people living with long COVID.
They found significantly lower levels of a marker that reflects the health of dopamine-producing systems across key regions of the brain compared with healthy participants.
The researchers discovered that reductions in dopamine markers were closely tied to patients' symptoms. Lower dopamine activity in one region of the brain, known as the ventral striatum, was associated with a greater loss of motivation.
Reduced dopamine markers in the dorsal putamen were linked to slower movement, while declines in the caudate putamen correlated with memory problems.
"Our findings provide compelling evidence that long COVID involves the loss of dopamine-releasing neurons," said senior author Dr. Jeffrey Meyer. "We know that inflammation can injure dopamine neurons. While our earlier research showed high levels of inflammation in those regions, this study provides direct evidence that the dopamine neuron marker is reduced in the same regions, and that this loss correlates with patients' symptoms."
Long COVID affects an estimated five percent of the global population and continues to leave millions struggling with persistent symptoms for months or even years after recovering from the initial infection.
Despite its widespread impact, there are currently no evidence-based treatments specifically approved for the condition because its underlying biological mechanisms remain poorly understood.
According to Dr. Meyer, the findings could mark a turning point in long COVID research. "These results indicate that long COVID is, at least in part, a disorder of the brain's dopamine system," he said. "This suggests that repurposing medications that augment the function of dopamine-releasing neurons, including dopamine precursors and inhibitors of dopamine metabolism, could be a promising approach."
The research also provides validation for many patients who have struggled to have their symptoms recognized.
"For five years I have been seeking answers on what happened to me after I contracted COVID in 2021," said Susan Deuville, a lived-experience research advisor involved in the project. "It was a crushing loss of the life I had and the person I was before. The research of Dr. Meyer brings hope. It also validates what long COVID sufferers have always known, long COVID is real and the effects are devastating."
The team plans to launch a clinical trial in the coming months to test whether existing medications that enhance dopamine function can improve memory, fatigue, and motivation in people with long COVID.
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