Liquor Impacting Brain Activity (Credit-Freepik)
Many of us believe that we are great drinkers and that alcohol does not affect us as much. People who are able to drink without showing any sign of inebriation are known as social drinkers. In short, they are not addicted to alcohol but will not turn down the opportunity to have a good time! While it may seem like it doesn’t affect you, new studies suggest that it is just an illusion, even if you have high tolerance, alcohol affects your cognitive and motor functions more than you think.
The study reveals the below implications and techniques:
Think of it as the foundation for your brain's performance. When brain conductivity is high, information flows smoothly, and that helps your brain in rapid processing and response. On the other hand, low conductivity can hinder cognitive function, leading to slower thinking, impaired memory, and difficulties with coordination.
A study conducted at the Neuroscience Research Australia (NeuRA) and UNSW Science unveiled a startling connection between alcohol consumption and brain conductivity.
While many people brush off the effects of alcohol as temporary changes in behaviour, the reality is much more complex. Beyond the obvious impacts on coordination and judgment, alcohol significantly alters brain function. Alcohol dramatically slowed down brain activity, especially in areas responsible for decision-making, planning, and physical coordination. This decline was so significant that it resembled the brain changes seen in normal ageing. This means even one drink could temporarily accelerate the ageing process of your brain.
The implications of this research are far-reaching. It provides compelling evidence that alcohol consumption has a direct and measurable impact on brain function. The discovery that alcohol can significantly reduce brain conductivity opens new avenues for understanding the neurocognitive effects of alcohol abuse and dependence. While you may not feel like alcohol is affecting you and you have a high tolerance, it most definitely changes and affects your decision-making abilities and impulse control.
Furthermore, the MRI technique employed in the study could be a valuable tool for assessing the impact of other substances on the brain and for developing interventions to mitigate alcohol-related brain damage.
Credit: iStock
One of the most effective ways to prevent rabies deaths could be by vaccinating dogs rather than just people before they are bitten. A new study published in The Lancet Regional Health – Southeast Asia modelled different strategies in Kerala for eradicating human rabies deaths between 2026 and 2035.
The study found that mass vaccination of dogs could bring the annual rabies deaths among people in Kerala close to zero within about three years even if 70% of the dog population were vaccinated.
On the other hand, vaccinating children before exposure to rabies was projected to prevent relatively few additional deaths at substantially higher cost.
India carries one of the worst largest rabies burdens. That's why the study could point towards a feasible and practical solution to eradicate the deadly infection.
Most human rabies deaths in India are linked to bites from infected dogs. This means that the disease can be tackled at its source by reducing transmission among dogs.
India records an estimated 9.1 million animal bites every year, with more than three-quarters caused by dogs. A 2024 estimate from the Indian Council of Medical Research's National Institute of Epidemiology estimates India's annual human rabies deaths to be about 5,726, which is an alarming public health issue.
The study puts emphasis on the fact that vaccinating a person after a bite protects that individual, whereas, vaccinating dogs reduces the probability that an infected dog will transmit rabies to many people in the first place.
Researchers from the University of Glasgow, Kerala's health and animal-husbandry departments, Kerala University of Health Sciences and the London School of Economics and Political Science used mathematical modelling to compare different strategies to combat rabies in Kerala.
One strategy was routine pre-exposure prophylaxis (PrEP) for children. Another focused on mass dog vaccination, while the model also considered improvements to post-exposure prophylaxis (PEP), the treatment given after a potentially rabid bite.
The researchers found that adding routine childhood PrEP provided minimal additional protection, preventing fewer than 10 deaths on average over a decade, while costing considerably more than strategies focused on controlling rabies transmission among dogs.
The model estimated that achieving 70% vaccination coverage among dogs could reduce annual human rabies deaths in Kerala to near zero within approximately three years.
Also read: Abhayrab Rabies Vaccine Batch Found Misbranded, ‘Not Of Standard Quality’: DCGI Alerts States
One of the study's striking findings was about the size of Kerala's stray-dog population. The researchers' modelling suggested that the state's actual stray-dog population could be around five times higher than official census estimates.
If vaccination programmes are based on an underestimated dog population, authorities could believe they have reached adequate coverage while a large proportion of dogs remain unvaccinated. That creates what epidemiologists call an immunity gap, allowing rabies transmission to continue.
Additionally, dog vaccination does not make human post-exposure treatment unnecessary. If a person is bitten or scratched by an animal that could have rabies, the wound should be washed thoroughly with soap and running water and medical care should be sought immediately.
Depending on the exposure, PEP can include rabies vaccine and rabies immunoglobulin (RIG). This is particularly important because rabies becomes extremely difficult to treat once symptoms begin.
The study also found that improving timely access to PEP could prevent more deaths than expanding routine pre-exposure vaccination of children.
Credit: X
The death of Lady Joan Branson, wife of billionaire entrepreneur Sir Richard Branson, has highlighted the risk of a blood clot after fractures that travels to the lungs.
Lady Branson, 80, died at Lister Hospital in London in November 2025 after being admitted following a fall at her home on Necker Island. An inquest heard that she had developed a deep vein thrombosis (DVT) and subsequently suffered a massive pulmonary embolism (PE).
The coroner, Professor Fiona Wilcox, concluded that her death could probably have been prevented if anti-clotting treatment had been given earlier. She said: “If this lady had been given [anti-clotting medication] early on ... her death, on the balance of probability, would have been prevented.”
What exactly happens after a fracture that can make a blood clot so dangerous?
A fracture can trigger several conditions that encourage the development of venous thromboembolism (VTE), the collective term for DVT and PE.
First, injury itself activates the body's clotting system. The body naturally increases clot formation after tissue damage to prevent bleeding.
Second, a person with a fracture may become significantly less mobile. When the leg muscles are not moving normally, the muscle pump that helps push blood through the veins becomes less effective. Blood can therefore pool in the deep veins, particularly in the legs. Hospitalisation, surgery, older age, previous blood clots and certain medical conditions can add further risk.
A clot that remains in a leg vein can cause swelling and pain. But when part of it breaks and gets away, it can travel through the bloodstream to the lungs, leading to a pulmonary embolism.
Also read: 9/11 Toxic Air Exposed Thousands: What New Records Reveal 25 Years Later
A clot forming in a deep vein, usually in the leg, can detach and travel through the veins into the right side of the heart. From there, it can enter the pulmonary arteries, which carry blood to the lungs.
If the clot is large enough, it can obstruct blood flow through the lungs and suddenly increase pressure on the right side of the heart.
A large or massive PE can therefore cause severe breathlessness, low blood pressure, cardiac arrest and sudden death.
This is why PE can sometimes appear dramatically, even when the original DVT produced few obvious symptoms.
According to the inquest, Lady Branson had a history of blood clots dating back to 2010, and doctors heard evidence that she had previously used anti-clotting injections.
However, she was not given enoxaparin, a low-molecular-weight heparin commonly used to prevent or treat harmful blood clots, during her hospital stay.
One doctor, Dr Inaki Bovill, told the inquest that a venous thromboembolism risk assessment should have been carried out and that the medication should have been prescribed. He said: “It is to my great regret that I did not.”
Another doctor, Dr George Adams, a consultant haematologist at Imperial College London, told the inquest that earlier anticoagulation would have made cardiac arrest less likely in her particular case.
Lady Branson's daughter, Dr Holly Branson, disputed the suggestion that her mother had refused the injections because of previous pain or bruising. She told the inquest that this was “not consistent with my knowledge of her usual practice or attitude.”
A DVT can cause:
A PE may cause:
Credit: AI
The US Food and Drug Administration (FDA) is investigating an outbreak of Salmonella Bovismorbificans infections in multiple states. According to reports, the at least infection cases are being linked to recalled broccoli sprouts across four states, including two hospitalisations. The outbreak hasn't resulted in any casualties yet.
The outbreak has been linked to broccoli sprouts grown by Evergreen Fresh Sprouts, LLC, based in Moyie Springs, Idaho. The FDA and the US Centers for Disease Control and Prevention (CDC) are investigating the outbreak with state and local health authorities.
The FDA has issued a clear warning: “Do not eat, serve, or sell recalled broccoli sprouts grown by Evergreen Fresh Sprouts, LLC.”
Also read: Granola Marketed For Nursing Mothers In US Recalled Over Potential Salmonella Contamination
According to the FDA, illnesses began between July 7 and August 26, 2026. Out of the 19 infected people who were interviewed by investigators, all 19 reported eating sprouts, while 17 specifically reported eating broccoli sprouts. That epidemiological pattern helped investigators identify sprouts as the likely driver of the infection.
Traceback investigations by the FDA and state partners then identified Evergreen Fresh Sprouts as the common grower of broccoli sprouts eaten by patients before they became sick.
On September 4, the company initiated a recall after Montana state officials found Salmonella in a sample of its broccoli sprout. Whole-genome sequencing showed that the Salmonella found in the product was the same strain causing illnesses in the outbreak.
The recalled products were sold in plastic bags or plastic clamshell containers and have expiration dates of September 7, 9, 11, 14 or 16, 2026.
Distribution has been confirmed in Idaho, Montana and Washington, although the FDA said the products may have reached additional states. The 22 cases are from Idaho, Montana, Utah and Washington.
Also read: Cyclospora to Alzheimer’s: Trump Administration Scales Back Key Health Research
Salmonella is a group of bacteria that can cause salmonellosis, a gastrointestinal infection usually acquired by consuming contaminated food or water.
Symptoms typically begin 12 to 72 hours after exposure and can include diarrhoea, fever and abdominal cramps. Most people recover within four to seven days without specific treatment, but some infections can become severe.
Young children, older adults and people with weakened immune systems are at greater risk of serious complications from the infection. In severe cases, Salmonella can cause dehydration or allow the bacteria to enter the bloodstream, potentially leading to invasive infection and complications affecting other organs.
Sprouts are grown in warm, moist conditions, which help seeds germinate but can also provide favourable conditions for bacterial growth.
The seeds themselves can become contaminated before sprouting. Once bacteria are present, the warm and humid environment used to produce sprouts can allow them to multiply. Since sprouts are often eaten raw, there may be no cooking step to eliminate the bacteria.
The current FDA investigation involves broccoli sprouts, and it is separate from another ongoing US investigation involving Salmonella and E. coli infections linked to alfalfa sprouts.
Consumers, restaurants and retailers should not eat, sell or serve the recalled Evergreen Fresh Sprouts broccoli sprouts.
The FDA also advises people who purchased or received the recalled product to carefully clean and sanitise surfaces and containers that may have come into contact with the sprouts. This is important because Salmonella can spread through cross-contamination.
Anyone who develops symptoms resembling Salmonella infection after eating the recalled sprouts should contact a healthcare provider.
© $2026 Times Horizon Private Limited